Mustafa M Siddiq
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Explore the profile of Mustafa M Siddiq including associated specialties, affiliations and a list of published articles.
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22
Citations
139
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Recent Articles
11.
Toro C, Hansen J, Siddiq M, Johnson K, Zhao W, Azulai D, et al.
Front Cell Neurosci
. 2021 Mar;
15:626192.
PMID: 33679326
Spinal cord injury (SCI) is a devastating form of neurotrauma. Patients who carry one or two apolipoprotein E (ApoE)4 alleles show worse functional outcomes and longer hospital stays after SCI,...
12.
Siddiq M, Chan A, Miorin L, Yadaw A, Beaumont K, Kehrer T, et al.
medRxiv
. 2020 Nov;
PMID: 33200140
COVID-19 affects multiple organs. Clinical data from the Mount Sinai Health System shows that substantial numbers of COVID-19 patients without prior heart disease develop cardiac dysfunction. How COVID-19 patients develop...
13.
Nikulina E, Gkioka V, Siddiq M, Mellado W, Hilaire M, Cain C, et al.
FEBS Lett
. 2020 Jan;
594(9):1389-1402.
PMID: 31985825
Rap1 is a small GTPase that has been implicated in dendritic development and plasticity. In this study, we investigated the role of Rap1 in axonal growth and its activation in...
14.
Yadaw A, Siddiq M, Rabinovich V, Tolentino R, Hansen J, Iyengar R
PLoS Comput Biol
. 2019 May;
15(5):e1006877.
PMID: 31042702
Whole cell responses involve multiple subcellular processes (SCPs). To understand how balance between SCPs controls the dynamics of whole cell responses we studied neurite outgrowth in rat primary cortical neurons...
15.
He H, Deng K, Siddiq M, Pyie A, Mellado W, Hannila S, et al.
J Neurosci
. 2016 Mar;
36(10):3079-91.
PMID: 26961960
Inhibitory molecules associated with CNS myelin, such as myelin-associated glycoprotein (MAG), represent major obstacles to axonal regeneration following CNS injury. Our laboratory has shown that elevating levels of intracellular cAMP,...
16.
Siddiq M, Hannila S
Front Mol Neurosci
. 2015 Jul;
8:26.
PMID: 26150769
Elevation of intracellular cyclic AMP (cAMP) levels has proven to be one of the most effective means of overcoming inhibition of axonal regeneration by myelin-associated inhibitors such as myelin-associated glycoprotein...
17.
Siddiq M, Hannila S, Carmel J, Bryson J, Hou J, Nikulina E, et al.
J Biol Chem
. 2015 May;
290(26):16343-56.
PMID: 25947372
The adult CNS does not spontaneously regenerate after injury, due in large part to myelin-associated inhibitors such as myelin-associated glycoprotein (MAG), Nogo-A, and oligodendrocyte-myelin glycoprotein. All three inhibitors can interact...
18.
Hannila S, Siddiq M, Carmel J, Hou J, Chaudhry N, Bradley P, et al.
J Neurosci
. 2013 Mar;
33(12):5138-51.
PMID: 23516280
After CNS injury, axonal regeneration is limited by myelin-associated inhibitors; however, this can be overcome through elevation of intracellular cyclic AMP (cAMP), as occurs with conditioning lesions of the sciatic...
19.
Emmetsberger J, Mirrione M, Zhou C, Fernandez-Monreal M, Siddiq M, Ji K, et al.
J Neurosci
. 2010 May;
30(19):6538-47.
PMID: 20463217
Glutamatergic neurons contain free zinc packaged into neurotransmitter-loaded synaptic vesicles. Upon neuronal activation, the vesicular contents are released into the synaptic space, whereby the zinc modulates activity of postsynaptic neurons...
20.
Hannila S, Siddiq M, Filbin M
Int Rev Neurobiol
. 2006 Dec;
77:57-105.
PMID: 17178472
No abstract available.