Kevin M Nickerson
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Explore the profile of Kevin M Nickerson including associated specialties, affiliations and a list of published articles.
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16
Citations
571
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Recent Articles
11.
Sweet R, Nickerson K, Cullen J, Wang Y, Shlomchik M
J Immunol
. 2017 Jun;
199(3):885-893.
PMID: 28659358
MyD88 and FcR common γ-chain (Fcer1g, FcRγ) elicit proinflammatory responses to exogenous Ags. Deletion of these receptors in autoimmune models has generally led to reduced overall disease. In B cells,...
12.
Gordon R, Herter J, Rosetti F, Campbell A, Nishi H, Kashgarian M, et al.
JCI Insight
. 2017 May;
2(10).
PMID: 28515361
Though recent reports suggest that neutrophil extracellular traps (NETs) are a source of antigenic nucleic acids in systemic lupus erythematosus (SLE), we recently showed that inhibition of NETs by targeting...
13.
Nickerson K, Wang Y, Bastacky S, Shlomchik M
PLoS One
. 2017 Mar;
12(3):e0173471.
PMID: 28278279
Genetic deficiency in TLR9 accelerates pathogenesis in the spontaneous polygenic MRL.Faslpr murine model of systemic lupus erythematosus, despite the absence of anti-nucleosome autoantibodies. However, it could be argued that this...
14.
Nickerson K, Cullen J, Kashgarian M, Shlomchik M
J Immunol
. 2013 Mar;
190(8):3889-94.
PMID: 23467932
TLR9 suppresses TLR7-driven pathogenesis in the MRL.Fas(lpr) murine model of systemic lupus erythematosus, but the mechanisms by which TLR7 promotes and TLR9 prevents disease in this and other lupus models...
15.
Nickerson K, Christensen S, Cullen J, Meng W, Luning Prak E, Shlomchik M
J Immunol
. 2013 Jan;
190(4):1447-56.
PMID: 23296704
Nucleic acid-reactive B cells frequently arise in the bone marrow but are tolerized by mechanisms including receptor editing, functional anergy, and/or deletion. TLR9, a sensor of endosomal dsDNA, both promotes...
16.
Nickerson K, Christensen S, Shupe J, Kashgarian M, Kim D, Elkon K, et al.
J Immunol
. 2010 Jan;
184(4):1840-8.
PMID: 20089701
Systemic lupus erythematosus is characterized by the production of autoantibodies against nucleic acid-associated Ags. We previously found that Tlr7 was required for anti-Sm and Tlr9 for anti-chromatin autoantibodies. Yet, although...