Stephen D Ginsberg
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Explore the profile of Stephen D Ginsberg including associated specialties, affiliations and a list of published articles.
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153
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5311
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Recent Articles
1.
Alldred M, Ibrahim K, Pidikiti H, Lee S, Heguy A, Chiosis G, et al.
Front Mol Neurosci
. 2025 Mar;
18:1546375.
PMID: 40078964
Introduction: Individuals with Down syndrome (DS) exhibit neurological deficits throughout life including the development of in Alzheimer's disease (AD) pathology and cognitive impairment. At the cellular level, dysregulation in neuronal...
2.
Counts S, Beck J, Maloney B, Malek-Ahmadi M, Ginsberg S, Mufson E, et al.
Alzheimers Dement
. 2025 Feb;
21(2):e70019.
PMID: 40008917
Introduction: MicroRNA (miRNA) activity is increasingly appreciated as a key regulator of pathophysiologic pathways in Alzheimer's disease (AD). However, the role of miRNAs during the progression of AD, including resilience...
3.
Bay S, Rodina A, Haut F, Roychowdhury T, Argyrousi E, Staniszewski A, et al.
Res Sq
. 2025 Feb;
PMID: 39989971
Alzheimer's disease (AD) progresses as a continuum, from preclinical stages to late-stage cognitive decline, yet the molecular mechanisms driving this progression remain poorly understood. Here, we provide a systems-level map...
4.
Alldred M, Ibrahim K, Pidikiti H, Chiosis G, Mufson E, Stutzmann G, et al.
Acta Neuropathol Commun
. 2024 Nov;
12(1):182.
PMID: 39605035
Selective vulnerability of neuronal populations occurs in both Down syndrome (DS) and Alzheimer's disease (AD), resulting in disproportional degeneration of pyramidal neurons (PNs) affecting memory and executive function. Elucidating the...
5.
Zhu B, Park J, Coffey S, Russo A, Hsu I, Wang J, et al.
Sci Transl Med
. 2024 Oct;
16(771):eabo1997.
PMID: 39475571
Parkinson's disease (PD) is a prevalent neurodegenerative disorder, and recent evidence suggests that pathogenesis may be in part mediated by inflammatory processes, the molecular and cellular architectures of which are...
6.
Roychowdhury T, McNutt S, Pasala C, Nguyen H, Thornton D, Sharma S, et al.
Nat Commun
. 2024 Oct;
15(1):8912.
PMID: 39414766
The intricate network of protein-chaperone interactions is crucial for maintaining cellular function. Recent discoveries have unveiled the existence of specialized chaperone assemblies, known as epichaperomes, which serve as scaffolding platforms...
7.
Gautier M, Kelley C, Lee S, Mufson E, Ginsberg S
Neurobiol Aging
. 2024 Sep;
144:30-42.
PMID: 39265450
Individuals with DS develop Alzheimer's disease (AD) neuropathology, including endosomal-lysosomal system abnormalities and degeneration of basal forebrain cholinergic neurons (BFCNs). We investigated whether maternal choline supplementation (MCS) affects early endosome...
8.
Ginsberg S, Blaser M
J Infect Dis
. 2024 Sep;
230(Supplement_2):S141-S149.
PMID: 39255394
Alzheimer's disease (AD) is a neurodegenerative disorder with limited therapeutic options. Accordingly, new approaches for prevention and treatment are needed. One focus is the human microbiome, the consortium of microorganisms...
9.
Alldred M, Pidikiti H, Ibrahim K, Lee S, Heguy A, Hoffman G, et al.
Acta Neuropathol
. 2024 Aug;
148(1):16.
PMID: 39105932
We elucidated the molecular fingerprint of vulnerable excitatory neurons within select cortical lamina of individuals with Down syndrome (DS) for mechanistic understanding and therapeutic potential that also informs Alzheimer's disease...
10.
Alldred M, Pidikiti H, Ibrahim K, Lee S, Heguy A, Hoffman G, et al.
J Alzheimers Dis
. 2024 Jul;
100(s1):S341-S362.
PMID: 39031371
Background: Individuals with Down syndrome (DS) have intellectual disability and develop Alzheimer's disease (AD) pathology during midlife, particularly in the hippocampal component of the medial temporal lobe memory circuit. However,...