Colleen M Croniger
Overview
Explore the profile of Colleen M Croniger including associated specialties, affiliations and a list of published articles.
Author names and details appear as published. Due to indexing inconsistencies, multiple individuals may share a name, and a single author may have variations. MedLuna displays this data as publicly available, without modification or verification
Snapshot
Snapshot
Articles
44
Citations
1900
Followers
0
Related Specialties
Related Specialties
Top 10 Co-Authors
Top 10 Co-Authors
Published In
Affiliations
Affiliations
Soon will be listed here.
Recent Articles
21.
Derecka M, Gornicka A, Koralov S, Szczepanek K, Morgan M, Raje V, et al.
Cell Metab
. 2012 Dec;
16(6):814-24.
PMID: 23217260
Mice lacking the Jak tyrosine kinase member Tyk2 become progressively obese due to aberrant development of Myf5+ brown adipose tissue (BAT). Tyk2 RNA levels in BAT and skeletal muscle, which...
22.
Berry D, Croniger C, Ghyselinck N, Noy N
Mol Cell Biol
. 2012 Jul;
32(19):3851-9.
PMID: 22826435
Vitamin A is secreted from cellular stores and circulates in blood bound to retinol-binding protein (RBP). In turn, holo-RBP associates in plasma with transthyretin (TTR) to form a ternary RBP-retinol-TTR...
23.
Buchner D, Geisinger J, Glazebrook P, Morgan M, Spiezio S, Kaiyala K, et al.
Mamm Genome
. 2012 Jul;
23(7-8):431-42.
PMID: 22752552
Despite considerable effort, the identification of genes that regulate complex multigenic traits such as obesity has proven difficult with conventional methodologies. The use of a chromosome substitution strain-based mapping strategy...
24.
Retinoic acid upregulates preadipocyte genes to block adipogenesis and suppress diet-induced obesity
Berry D, DeSantis D, Soltanian H, Croniger C, Noy N
Diabetes
. 2012 Mar;
61(5):1112-21.
PMID: 22396202
Retinoic acid (RA) protects mice from diet-induced obesity. The activity is mediated in part through activation of the nuclear receptors RA receptors (RARs) and peroxisome proliferator-activated receptor β/δ and their...
25.
Ye F, Lemieux H, Hoppel C, Hanson R, Hakimi P, Croniger C, et al.
J Biol Chem
. 2011 Sep;
286(46):40013-24.
PMID: 21917928
Overexpression of the Ski oncogene induces oncogenic transformation of chicken embryo fibroblasts (CEFs). However, unlike most other oncogene-transformed cells, Ski-transformed CEFs (Ski-CEFs) do not display the classical Warburg effect. On...
26.
Yazbek S, Buchner D, Geisinger J, Burrage L, Spiezio S, Zentner G, et al.
Genome Res
. 2011 Apr;
21(7):1065-73.
PMID: 21507882
Although central to many studies of phenotypic variation and disease susceptibility, characterizing the genetic architecture of complex traits has been unexpectedly difficult. For example, most of the susceptibility genes that...
27.
Hsieh C, Huang C, Bederman I, Yang J, Beidelschies M, Hatzoglou M, et al.
J Lipid Res
. 2011 Apr;
52(7):1352-62.
PMID: 21504969
Previously, we have shown that Pck1 expression in mammary gland adipocytes and white adipose tissue maintains triglyceride stores through glyceroneogenesis, and these lipids were used for synthesis of milk triglycerides...
28.
Christopher B, Huang H, Berthiaume J, McElfresh T, Chen X, Croniger C, et al.
Am J Physiol Heart Circ Physiol
. 2010 Sep;
299(6):H1917-27.
PMID: 20852054
Previous studies have reported that high fat feeding in mild to moderate heart failure (HF) results in the preservation of contractile function. Recent evidence has suggested that preventing the switch...
29.
Ayala J, Samuel V, Morton G, Obici S, Croniger C, Shulman G, et al.
Dis Model Mech
. 2010 Aug;
3(9-10):525-34.
PMID: 20713647
The Mouse Metabolic Phenotyping Center (MMPC) Consortium was established to address the need to characterize the growing number of mouse models of metabolic diseases, particularly diabetes and obesity. A goal...
30.
Burrage L, Baskin-Hill A, Sinasac D, Singer J, Croniger C, Kirby A, et al.
Mamm Genome
. 2010 Feb;
21(3-4):115-29.
PMID: 20127486
Discovery of genes that confer resistance to diseases such as diet-induced obesity could have tremendous therapeutic impact. We previously demonstrated that the C57BL/6J-Chr(A/J)/NaJ panel of chromosome substitution strains (CSSs) is...