Wen Qing Li
Overview
Explore the profile of Wen Qing Li including associated specialties, affiliations and a list of published articles.
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20
Citations
935
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Recent Articles
11.
Liacini A, Sylvester J, Li W, Zafarullah M
Arthritis Res Ther
. 2005 Jul;
7(4):R777-83.
PMID: 15987479
Interleukin-1 (IL-1), IL-17 and tumor necrosis factor alpha (TNF-alpha) are the main proinflammatory cytokines implicated in cartilage breakdown by matrix metalloproteinase (MMPs) in arthritic joints. We studied the impact of...
12.
Khaled A, Bulavin D, Kittipatarin C, Li W, Alvarez M, Kim K, et al.
J Cell Biol
. 2005 Jun;
169(5):755-63.
PMID: 15928203
Lymphocytes are the central mediators of the immune response, requiring cytokines for survival and proliferation. Survival signaling targets the Bcl-2 family of apoptotic mediators, however, the pathway for the cytokine-driven...
13.
Jiang Q, Li W, Hofmeister R, Young H, Hodge D, Keller J, et al.
Mol Cell Biol
. 2004 Jul;
24(14):6501-13.
PMID: 15226449
The antiapoptotic function of the interleukin-7 (IL-7) receptor is related to regulation of three members of the Bcl2 family: synthesis of Bcl2, phosphorylation of Bad, and cytosolic retention of Bax....
14.
Li W, Qureshi H, Liacini A, Dehnade F, Zafarullah M
Free Radic Biol Med
. 2004 Jun;
37(2):196-207.
PMID: 15203191
Transforming growth factor beta1 (TGF-beta1) stimulates cartilage extracellular matrix synthesis but, in excess, evokes synovial inflammation, hyperplasia, and osteophyte formation in arthritic joints. TGF-beta1 induces tissue inhibitor of metalloproteinases 3...
15.
Li W, Jiang Q, Khaled A, Keller J, Durum S
J Biol Chem
. 2004 May;
279(28):29160-6.
PMID: 15123689
Interleukin-7 (IL-7) is a cytokine that is required for T cell development and survival. The anti-apoptotic function of IL-7 is partly through induction of Bcl-2 synthesis and cytosolic retention of...
16.
Sylvester J, Liacini A, Li W, Zafarullah M
Cell Signal
. 2004 Jan;
16(4):469-76.
PMID: 14709335
Interleukin (IL)-17 promotes cartilage breakdown by inducing matrix metalloproteinases (MMPs) and aggrecanases (a disintegrin and metalloproteinase with thrombospondin motif, ADAMTS) in arthritic joints. We investigated IL-17 signaling pathways inducing MMP-3,...
17.
Liacini A, Sylvester J, Li W, Huang W, Dehnade F, Ahmad M, et al.
Exp Cell Res
. 2003 Jul;
288(1):208-17.
PMID: 12878172
Tumor necrosis factor alpha (TNF-alpha), a major proinflammatory cytokine, induces arthritic joint inflammation and resorption of cartilage by matrix metalloproteinase-13 (MMP-13). RNA for MMP-13 is increased in human arthritic femoral...
18.
Khaled A, Li W, Huang J, Fry T, Khaled A, Mackall C, et al.
Immunity
. 2002 Nov;
17(5):561-73.
PMID: 12433363
The requirement for cytokines in hematopoiesis is partly attributable to the protection of cells from apoptosis. Since IL-7 is required for normal T cell development, we evaluated the role of...
19.
Liacini A, Sylvester J, Li W, Zafarullah M
Matrix Biol
. 2002 May;
21(3):251-62.
PMID: 12009331
Interleukin-1 (IL-1), the main cytokine instigator of cartilage degeneration in arthritis, induces matrix metalloproteinase-3 (MMP-3) and MMP-13 RNA and protein in chondrocytes. The molecular mechanisms of this induction were investigated...
20.
Huang W, Li W, Dehnade F, Zafarullah M
J Cell Biochem
. 2002 Apr;
85(2):295-303.
PMID: 11948685
Tissue inhibitor of metalloproteinases-4 (TIMP-4), the newest member of the TIMP family, blocks the activities of several matrix metalloproteinases (MMPs) implicated in the arthritic cartilage erosion. By utilizing semi-quantitative RT-PCR,...