Wen-Quan Zou
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Explore the profile of Wen-Quan Zou including associated specialties, affiliations and a list of published articles.
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82
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1803
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Recent Articles
1.
Parveen S, Alam P, Orru C, Vascellari S, Hughson A, Zou W, et al.
NPJ Biosens
. 2025 Feb;
2(1):8.
PMID: 39950033
Parkinson's disease (PD), dementia with Lewy bodies (DLB), and other synucleinopathies are characterized by the accumulation of abnormal, self-propagating aggregates of α-synuclein. RT-QuIC or seed amplification assays are currently showing...
2.
Wang Z, Wu L, Gerasimenko M, Gilliland T, Shah Z, Lomax E, et al.
Mol Neurodegener
. 2024 Nov;
19(1):92.
PMID: 39609917
Background: Tauopathies are a group of age-related neurodegenerative diseases characterized by the accumulation of pathologically hyperphosphorylated tau protein in the brain, leading to prion-like aggregation and propagation. They include Alzheimer's...
3.
Chen Z, Shi Q, Xiao K, Kong Y, Liang D, Wang Y, et al.
JAMA Neurol
. 2024 Oct;
81(12):1263-1273.
PMID: 39401015
Importance: Recent studies have revealed that autopsy skin samples from cadavers with prion diseases (PRDs) exhibited a positive prion seeding activity similar to cerebrospinal fluid (CSF). It is worthwhile to...
4.
Srivastava A, Wang Q, Orru C, Fernandez M, Compta Y, Ghetti B, et al.
PLoS Pathog
. 2024 Sep;
20(9):e1012554.
PMID: 39302978
Disease associated pathological aggregates of alpha-synuclein (αSynD) exhibit prion-like spreading in synucleinopathies such as Parkinson's disease (PD) and dementia with Lewy bodies (DLB). Seed amplification assays (SAAs) such as real-time...
5.
Mao H, Kuang Y, Feng D, Chen X, Lu L, Xia W, et al.
Transl Neurodegener
. 2024 Jul;
13(1):35.
PMID: 39049095
Background: Seed amplification assays (SAA) enable the amplification of pathological misfolded proteins, including α-synuclein (αSyn), in both tissue homogenates and body fluids of Parkinson's disease (PD) patients. SAA involves repeated...
6.
LoPiccolo M, Wang Z, Maayan Eshed G, Fierro L, Stauffer C, Wang K, et al.
Mov Disord
. 2024 Jul;
39(11):2087-2091.
PMID: 39021250
Background: Patients with type 1 Gaucher disease (GD1) have a significantly increased risk of developing Parkinson's disease (PD). Objective: The objective of this study was to evaluate skin α-synuclein (αSyn)...
7.
Jiang D, Nan H, Chen Z, Zou W, Wu L
Neurobiol Dis
. 2024 Jul;
199:106599.
PMID: 38996988
Objective: Sporadic Creutzfeldt-Jakob disease (sCJD) is a fatal rapidly progressive neurodegenerative disorder with no effective therapeutic interventions. We aimed to identify potential genetically-supported drug targets for sCJD by integrating multi-omics...
8.
Memon S, Wang Z, Zou W, Kim Y, Jeong B
Animals (Basel)
. 2024 Jun;
14(12).
PMID: 38929426
Prion disorders are fatal infectious diseases that are caused by a buildup of pathogenic prion protein (PrP) in susceptible mammals. According to new findings, the shadow of prion protein (Sho)...
9.
Chen Z, Kong Y, Zhang J, Zou W, Wu L
Neurobiol Dis
. 2024 Apr;
195:106497.
PMID: 38583641
Objectives: To elucidate and compare the genetic, clinical, ancillary diagnostic, and pathological characteristics across different Gerstmann-Sträussler-Scheinker disease (GSS) phenotypes and explore the underlying causes of the phenotypic heterogeneities. Methods: The...
10.
Wang Z, Wu L, Gerasimenko M, Gilliland T, Gunzler S, Donadio V, et al.
Res Sq
. 2024 Mar;
PMID: 38496453
Background: Tauopathies are a group of age-related neurodegenerative diseases characterized by the accumulation of pathologically phosphorylated tau protein in the brain, leading to prion-like propagation and aggregation. They include Alzheimer's...