Wen-Hao Hsu
Overview
Explore the profile of Wen-Hao Hsu including associated specialties, affiliations and a list of published articles.
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Articles
32
Citations
738
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Recent Articles
1.
Liu Y, Han J, Hsu W, LaBella K, Deng P, Shang X, et al.
Cancer Discov
. 2024 Sep;
15(1):162-178.
PMID: 39348506
Clinically available KRAS* inhibitors and IO agents alleviated the immunosuppressive tumor microenvironment in PDAC. Profound tumor regression and prolonged survival in an autochthonous PDAC model provide a compelling rationale for...
2.
Drainas A, Hsu W, Dallas A, Poltorack C, Kim J, He A, et al.
Cell Rep
. 2024 Aug;
43(8):114606.
PMID: 39120974
Patients with small-cell lung cancer (SCLC) are in dire need of more effective therapeutic options. Frequent disruption of the G1 checkpoint in SCLC cells creates a dependency on the G2/M...
3.
LaBella K, Hsu W, Li J, Qi Y, Liu Y, Liu J, et al.
Dev Cell
. 2024 Apr;
59(11):1475-1486.e5.
PMID: 38574731
Telomere dynamics are linked to aging hallmarks, and age-associated telomere loss fuels the development of epithelial cancers. In Apc-mutant mice, the onset of DNA damage associated with telomere dysfunction has...
4.
Khan F, Lin Y, Ali H, Pang L, Dunterman M, Hsu W, et al.
Nat Commun
. 2024 Mar;
15(1):1987.
PMID: 38443336
Abundant macrophage infiltration and altered tumor metabolism are two key hallmarks of glioblastoma. By screening a cluster of metabolic small-molecule compounds, we show that inhibiting glioblastoma cell glycolysis impairs macrophage...
5.
Khan F, Lin Y, Ali H, Pang L, Dunterman M, Hsu W, et al.
Res Sq
. 2023 Oct;
PMID: 37886538
Abundant macrophage infiltration and altered tumor metabolism are two key hallmarks of glioblastoma. By screening a cluster of metabolic small-molecule compounds, we show that inhibiting glioblastoma cell glycolysis impairs macrophage...
6.
Hsu W, LaBella K, Lin Y, Xu P, Lee R, Hsieh C, et al.
Cancer Discov
. 2023 Sep;
13(12):2652-2673.
PMID: 37768068
Significance: This study identified a molecular mechanism contributing to KRAS*-driven colorectal cancer progression via fibroblast transformation in the tumor microenvironment to produce VEGFA driving tumor angiogenesis. In preclinical models, targeting...
7.
Pang L, Dunterman M, Guo S, Khan F, Liu Y, Taefi E, et al.
Nat Immunol
. 2023 Sep;
24(10):1654-1670.
PMID: 37667051
Glioblastoma (GBM) tumors consist of multiple cell populations, including self-renewing glioblastoma stem cells (GSCs) and immunosuppressive microglia. Here we identified Kunitz-type protease inhibitor TFPI2 as a critical factor connecting these...
8.
Li J, Lan Z, Liao W, Horner J, Xu X, Liu J, et al.
Nature
. 2023 Jun;
619(7970):632-639.
PMID: 37344599
Sex exerts a profound impact on cancer incidence, spectrum and outcomes, yet the molecular and genetic bases of such sex differences are ill-defined and presumptively ascribed to X-chromosome genes and...
9.
Khadka S, Lin Y, Ackroyd J, Chen Y, Sheng Y, Qian W, et al.
bioRxiv
. 2023 May;
PMID: 37214825
Tumor angiogenesis is a cancer hallmark, and its therapeutic inhibition has provided meaningful, albeit limited, clinical benefit. While anti-angiogenesis inhibitors deprive the tumor of oxygen and essential nutrients, cancer cells...
10.
Pang L, Dunterman M, Xuan W, Gonzalez A, Lin Y, Hsu W, et al.
Cell Rep
. 2023 Feb;
42(2):112127.
PMID: 36795563
Glioblastoma (GBM) is one of the most aggressive tumors in the adult central nervous system. We previously revealed that circadian regulation of glioma stem cells (GSCs) affects GBM hallmarks of...