Stephen M Cohen
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Explore the profile of Stephen M Cohen including associated specialties, affiliations and a list of published articles.
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116
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9427
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Recent Articles
1.
Habermann A, Timmerman W, Cohen S, Burkhardt B, Amendola M
Int J Colorectal Dis
. 2024 Aug;
39(1):127.
PMID: 39107626
Background: The utilization of three-dimensional printing has grown rapidly within the field of surgery over recent years. Within the subspecialty of colorectal surgery, the technology has been used to create...
2.
Groth C, Vaid P, Khatpe A, Prashali N, Ahiya A, Andrejeva D, et al.
G3 (Bethesda)
. 2020 Aug;
10(9):2999-3008.
PMID: 32737065
Genetic approaches in have successfully identified many genes involved in regulation of growth control as well as genetic interactions relevant to the initiation and progression of cancer Here, we report...
3.
Song S, Andrejeva D, Freitas F, Cohen S, Herranz H
Proc Natl Acad Sci U S A
. 2019 Jun;
116(28):14055-14064.
PMID: 31235567
Wnt/Wingless (Wg) signaling controls many aspects of animal development and is deregulated in different human cancers. The transcription factor dTcf/Pangolin (Pan) is the final effector of the Wg pathway in...
4.
Hanyu-Nakamura K, Matsuda K, Cohen S, Nakamura A
Development
. 2019 Mar;
146(7).
PMID: 30890569
Specification of germ cells is pivotal to ensure continuation of animal species. In many animal embryos, germ cell specification depends on maternally supplied determinants in the germ plasm. () mRNA...
5.
Kirsch R, Seemann S, Ruzzo W, Cohen S, Stadler P, Gorodkin J
BMC Genomics
. 2018 Dec;
19(1):899.
PMID: 30537930
Background: Comparative genomics approaches have facilitated the discovery of many novel non-coding and structured RNAs (ncRNAs). The increasing availability of related genomes now makes it possible to systematically search for...
6.
Andrejeva D, Kugler J, Nguyen H, Malmendal A, Holm M, Toft B, et al.
BMC Cancer
. 2018 Nov;
18(1):1180.
PMID: 30486822
Background: Changes in cellular metabolism are now recognized as potential drivers of cancer development, rather than as secondary consequences of disease. Here, we explore the mechanism by which metabolic changes...
7.
Eichenlaub T, Villadsen R, Freitas F, Andrejeva D, Aldana B, Nguyen H, et al.
Curr Biol
. 2018 Oct;
28(20):3220-3228.e6.
PMID: 30293715
Cancers develop in a complex mutational landscape. Genetic models of tumor formation have been used to explore how combinations of mutations cooperate to promote tumor formation in vivo. Here, we...
8.
Herranz H, Cohen S
J Dev Biol
. 2018 Apr;
5(4).
PMID: 29615570
Cellular metabolism has recently been recognized as a hallmark of cancer. Investigating the origin and effects of the reprogrammed metabolism of tumor cells, and identifying its genetic mediators, will improve...
9.
Nguyen H, Kugler J, Loya A, Cohen S
Oncotarget
. 2017 Oct;
8(38):64095-64105.
PMID: 28969054
The Hippo pathway, which acts to repress the activity of YAP and TAZ trancriptional co-activators, serve as a barrier for oncogenic transformation. Unlike other oncoproteins, YAP and TAZ are rarely...
10.
Song S, Herranz H, Cohen S
Dis Model Mech
. 2017 Jul;
10(10):1201-1209.
PMID: 28754838
Switch/sucrose non-fermentable (SWI/SNF) chromatin remodeling complexes are mutated in many human cancers. In this article, we make use of a genetic model for epithelial tumor formation to explore the tumor...