Stephen D Hauschka
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Explore the profile of Stephen D Hauschka including associated specialties, affiliations and a list of published articles.
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46
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1627
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Recent Articles
11.
Tawara N, Yamashita S, Kawakami K, Kurashige T, Zhang Z, Tasaki M, et al.
Exp Neurol
. 2018 Aug;
309:169-180.
PMID: 30130494
Muscle histology of sporadic inclusion body myositis (sIBM) demonstrates inflammatory findings and degenerative features including accumulation of TAR DNA-binding protein of 43 kDa (TDP-43). However, whether sarcoplasmic accumulation of TDP-43...
12.
Amoasii L, Long C, Li H, Mireault A, Shelton J, Sanchez-Ortiz E, et al.
Sci Transl Med
. 2017 Dec;
9(418).
PMID: 29187645
Duchenne muscular dystrophy (DMD) is a severe, progressive muscle disease caused by mutations in the dystrophin gene. The majority of DMD mutations are deletions that prematurely terminate the dystrophin protein....
13.
Bengtsson N, Hall J, Odom G, Phelps M, Andrus C, Hawkins R, et al.
Nat Commun
. 2017 Jun;
8:16007.
PMID: 28643790
This corrects the article DOI: 10.1038/ncomms14454.
14.
Thomson K, Odom G, Murry C, Mahairas G, Moussavi-Harami F, Teichman S, et al.
JACC Basic Transl Sci
. 2017 May;
1(7):666-679.
PMID: 28553667
Despite recent advances, chronic heart failure remains a significant and growing unmet medical need, reaching epidemic proportions carrying substantial morbidity, mortality, and costs. A safe and convenient therapeutic agent that...
15.
Bengtsson N, Hall J, Odom G, Phelps M, Andrus C, Hawkins R, et al.
Nat Commun
. 2017 Feb;
8:14454.
PMID: 28195574
Gene replacement therapies utilizing adeno-associated viral (AAV) vectors hold great promise for treating Duchenne muscular dystrophy (DMD). A related approach uses AAV vectors to edit specific regions of the DMD...
16.
Kolwicz Jr S, Odom G, Nowakowski S, Moussavi-Harami F, Chen X, Reinecke H, et al.
Mol Ther
. 2015 Sep;
24(2):240-250.
PMID: 26388461
Impaired systolic function, resulting from acute injury or congenital defects, leads to cardiac complications and heart failure. Current therapies slow disease progression but do not rescue cardiac function. We previously...
17.
Muir L, Nguyen Q, Hauschka S, Chamberlain J
Mol Ther Methods Clin Dev
. 2015 Jan;
1:14025.
PMID: 25558461
Autologous dermal fibroblasts are promising candidates for enhancing muscle regeneration in Duchenne muscular dystrophy (DMD) due to their ease of isolation, immunological compatibility, and greater proliferative potential than DMD satellite...
18.
Hu C, Kasten J, Park H, Bhargava R, Tai D, Grody W, et al.
Mol Ther
. 2014 Jun;
22(10):1792-802.
PMID: 24888478
Human arginase deficiency is characterized by hyperargininemia and infrequent episodes of hyperammonemia that cause neurological impairment and growth retardation. We previously developed a neonatal mouse adeno-associated viral vector (AAV) rh10-mediated...
19.
White Moyes K, Sip C, Obenza W, Yang E, Horst C, Welikson R, et al.
Stem Cells Dev
. 2013 Mar;
22(16):2315-25.
PMID: 23517131
An improved understanding of the factors that regulate the migration of human embryonic stem cell-derived cardiomyocytes (hESC-CMs) would provide new insights into human heart development and suggest novel strategies to...
20.
Gantz J, Palpant N, Welikson R, Hauschka S, Murry C, Laflamme M
PLoS One
. 2012 Oct;
7(10):e46971.
PMID: 23071682
The differentiation of pluripotent stem cells involves transition through a series of specific cell states. To understand these cell fate decisions, the field needs improved genetic tools for the labeling,...