Stefan F Lichtenthaler
Overview
Explore the profile of Stefan F Lichtenthaler including associated specialties, affiliations and a list of published articles.
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Articles
187
Citations
5884
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Recent Articles
1.
Jocher G, Ozcelik G, Muller S, Hsia H, Lastra Osua M, Hofmann L, et al.
Life Sci Alliance
. 2025 Mar;
8(5).
PMID: 40081988
The cell surface receptor TREM2 is a key genetic risk factor and drug target in Alzheimer's disease (AD). In the brain, TREM2 is expressed in microglia, where it undergoes proteolytic...
2.
Takalo M, Jeskanen H, Rolova T, Kervinen I, Hellen M, Heikkinen S, et al.
J Neuroinflammation
. 2025 Mar;
22(1):64.
PMID: 40038760
Background: Phospholipase C gamma 2, proline 522 to arginine (PLCγ2-P522R) is a protective variant that reduces the risk of Alzheimer's disease (AD). Recently, it was shown to mitigate β-amyloid pathology...
3.
Kardell O, Gronauer T, von Toerne C, Merl-Pham J, Konig A, Barth T, et al.
J Proteome Res
. 2025 Feb;
24(3):1017-1029.
PMID: 39918541
Advancing MS-based proteomics toward clinical applications evolves around developing standardized start-to-finish and fit-for-purpose workflows for clinical specimens. Steps along the method design involve the determination and optimization of several bioanalytical...
4.
Guilbaud L, Roger K, Schmidt A, Chhuon C, Breimann S, Lipecka J, et al.
J Proteomics
. 2025 Jan;
313:105372.
PMID: 39778765
Despite numerous studies on fetal therapy for myelomeningoceles (MMC), the pathophysiology of this malformation remains poorly understood. This study aimed to analyze the biochemical profile and proteome of amniotic fluid...
5.
Marvian A, Strauss T, Tang Q, Tuck B, Keeling S, Rudiger D, et al.
Mol Neurodegener
. 2024 Dec;
19(1):100.
PMID: 39736627
Background: The prion-like spreading of Tau pathology is the leading cause of disease progression in various tauopathies. A critical step in propagating pathologic Tau in the brain is the transport...
6.
Dinkel L, Hummel S, Zenatti V, Malara M, Tillmann Y, Colombo A, et al.
Sci Transl Med
. 2024 Dec;
16(776):eadl4616.
PMID: 39630885
Niemann-Pick type C (NPC) disease is an inherited lysosomal storage disorder mainly driven by mutations in the gene, causing lipid accumulation within late endosomes/lysosomes and resulting in progressive neurodegeneration. Although...
7.
Yoshida F, Nagatomo R, Utsunomiya S, Kimura M, Shun S, Kono R, et al.
Transl Psychiatry
. 2024 Oct;
14(1):448.
PMID: 39443477
Autism Spectrum Disorder (ASD) is a developmental disorder characterized by impaired social communication and repetitive behaviors. In recent years, a pharmacological mouse model of ASD involving maternal administration of valproic...
8.
Yasa S, Butz E, Colombo A, Chandrachud U, Montore L, Tschirner S, et al.
Commun Biol
. 2024 Oct;
7(1):1373.
PMID: 39438652
Loss-of-function mutations in CLN3 cause juvenile Batten disease, featuring neurodegeneration and early-stage neuroinflammation. How loss of CLN3 function leads to early neuroinflammation is not yet understood. Here, we have comprehensively...
9.
Bonelli S, Lo Pinto M, Ye Y, Muller S, Lichtenthaler S, Scilabra S
Mol Cell Proteomics
. 2024 Oct;
23(11):100854.
PMID: 39389361
Ubiquitin carboxyl-terminal hydrolase 19 (USP19) is a unique deubiquitinase, characterized by multiple variants generated by alternative splicing. Several variants bear a C-terminal transmembrane domain that anchors them to the endoplasmic...
10.
Calligaris M, Spano D, Puccio M, Muller S, Bonelli S, Lo Pinto M, et al.
Proteomics
. 2024 Sep;
24(23-24):e202400076.
PMID: 39318062
Ectodomain shedding, which is the proteolytic release of transmembrane proteins from the cell surface, is crucial for cell-to-cell communication and other biological processes. The metalloproteinase ADAM17 mediates ectodomain shedding of...