Somanathapura K NaveenKumar
Overview
Explore the profile of Somanathapura K NaveenKumar including associated specialties, affiliations and a list of published articles.
Author names and details appear as published. Due to indexing inconsistencies, multiple individuals may share a name, and a single author may have variations. MedLuna displays this data as publicly available, without modification or verification
Snapshot
Snapshot
Articles
24
Citations
304
Followers
0
Related Specialties
Related Specialties
Top 10 Co-Authors
Top 10 Co-Authors
Published In
Published In
Affiliations
Affiliations
Soon will be listed here.
Recent Articles
1.
Manikanta K, NaveenKumar S, Hemshekhar M, Thushara R, Mugesh G, Kemparaju K, et al.
Phytomedicine
. 2025 Jan;
138:156386.
PMID: 39842372
Background: Cellular histones are DNA-binding nuclear proteins involved in chromatin remodelling and regulation of gene expression. However, extracellular histones act as damage-associated molecular patterns (DAMPs) and contribute to multiorgan damage...
2.
Manikanta , NaveenKumar S, Thushara R, Hemshekhar M, Sumedini M, Sunitha K, et al.
Thromb Res
. 2024 Oct;
244:109199.
PMID: 39467509
Platelets are essential for normal hemostasis and thrombosis but become hyperactive in hemolytic disorders. Cell-free heme is known to be toxic to platelets and endothelial cells, playing a significant role...
3.
Tambralli A, Harbaugh A, NaveenKumar S, Radyk M, Rysenga C, Sabb K, et al.
J Clin Invest
. 2024 Jun;
134(15).
PMID: 38869951
Neutrophil hyperactivity and neutrophil extracellular trap release (NETosis) appear to play important roles in the pathogenesis of the thromboinflammatory autoimmune disease known as antiphospholipid syndrome (APS). The understanding of neutrophil...
4.
Sloan E, Kmetova K, NaveenKumar S, Kluge L, Chong E, Hoy C, et al.
Clin Immunol
. 2024 Feb;
261:109926.
PMID: 38355030
Our study aimed to evaluate the presence, clinical associations, and potential mechanistic roles of non-criteria antiphospholipid antibodies (aPL) and circulating calprotectin, a highly stable marker of neutrophil extracellular trap release...
5.
NaveenKumar S, Tambralli A, Fonseca B, Yalavarthi S, Liang W, Hoy C, et al.
Blood
. 2024 Jan;
143(12):1193-1197.
PMID: 38237140
Many patients with antiphospholipid syndrome had decreased ectonucleotidase activity on neutrophils and platelets, which enabled extracellular nucleotides to trigger neutrophil-platelet aggregates. This phenotype was replicated by treating healthy neutrophils and...
6.
Hoy C, NaveenKumar S, Navaz S, Sugur K, Yalavarthi S, Sarosh C, et al.
Arthritis Rheumatol
. 2024 Jan;
76(6):928-935.
PMID: 38225923
Objective: While thrombosis and pregnancy loss are the best-known clinical features of antiphospholipid syndrome (APS), many patients also exhibit "extra-criteria" manifestations, such as thrombocytopenia. The mechanisms that drive APS thrombocytopenia...
7.
Liu C, Yalavarthi S, Tambralli A, Zeng L, Rysenga C, Alizadeh N, et al.
Sci Adv
. 2023 Oct;
9(43):eadj1019.
PMID: 37878711
While neutrophil extracellular traps (NETs) have previously been linked to some diabetes-associated complications, such as dysfunctional wound healing, their potential role in diabetic vascular dysfunction has not been studied. Diabetic...
8.
NaveenKumar S, Hemshekhar M, Sharathbabu B, Kemparaju K, Mugesh G, Girish K
Biochim Biophys Acta Mol Basis Dis
. 2023 Mar;
1869(5):166688.
PMID: 36925054
Cell-free heme (CFH) is a product of hemoglobin, myoglobin and hemoprotein degradation, which is a hallmark of pathologies associated with extensive hemolysis and tissue damage. CHF and iron collectively induce...
9.
NaveenKumar S, Knight J
Nat Rev Rheumatol
. 2022 Sep;
18(12):675-676.
PMID: 36180549
No abstract available.
10.
Singh N, NaveenKumar S, Geethika M, Mugesh G
Angew Chem Int Ed Engl
. 2020 Oct;
60(6):3121-3130.
PMID: 33079465
Nanoparticles that functionally mimic the activity of metal-containing enzymes (metallo-nanozymes) are of therapeutic importance for treating various diseases. However, it is still not clear whether such nanozymes can completely substitute...