Robert A J Byrne
Overview
Explore the profile of Robert A J Byrne including associated specialties, affiliations and a list of published articles.
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Articles
4
Citations
31
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0
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Recent Articles
1.
Carter-Cusack D, Huang S, Keshvari S, Patkar O, Sehgal A, Allavena R, et al.
PLoS Genet
. 2025 Jan;
21(1):e1011525.
PMID: 39869647
Adaptation to existence outside the womb is a key event in the life of a mammal. The absence of macrophages in rats with a homozygous mutation in the colony-stimulating factor...
2.
Nimmo J, Byrne R, Daskoulidou N, Watkins L, Carpanini S, Zelek W, et al.
Clin Sci (Lond)
. 2024 Mar;
138(6):387-412.
PMID: 38505993
Complement is an important component of innate immune defence against pathogens and crucial for efficient immune complex disposal. These core protective activities are dependent in large part on properly regulated...
3.
Carpanini S, Torvell M, Bevan R, Byrne R, Daskoulidou N, Saito T, et al.
Acta Neuropathol Commun
. 2022 Jul;
10(1):99.
PMID: 35794654
Complement is involved in developmental synaptic pruning and pathological synapse loss in Alzheimer's disease. It is posited that C1 binding initiates complement activation on synapses; C3 fragments then tag them...
4.
Torvell M, Carpanini S, Daskoulidou N, Byrne R, Sims R, Morgan B
Genes (Basel)
. 2021 Dec;
12(12).
PMID: 34946939
The presence of complement activation products at sites of pathology in post-mortem Alzheimer's disease (AD) brains is well known. Recent evidence from genome-wide association studies (GWAS), combined with the demonstration...
5.
Byrne R, Torvell M, Daskoulidou N, Fathalla D, Kokkali E, Carpanini S, et al.
Mol Neurobiol
. 2021 May;
58(9):4323-4336.
PMID: 34002346
Recent studies have identified roles for complement in synaptic pruning, both physiological during development and pathological in Alzheimer's disease (AD). These reports suggest that C1q initiates complement activation on synapses...