Paula M Oliver
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Explore the profile of Paula M Oliver including associated specialties, affiliations and a list of published articles.
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36
Citations
972
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Recent Articles
1.
Dar A, Ortega Y, Aktas S, Wu K, Guha I, Porter N, et al.
J Immunol
. 2024 Jan;
212(6):982-991.
PMID: 38265261
Multiple sclerosis, and its murine model experimental autoimmune encephalomyelitis (EAE), is a neurodegenerative autoimmune disease of the CNS characterized by T cell influx and demyelination. Similar to other autoimmune diseases,...
2.
Wang Y, Manzi M, Feswick A, Renshaw L, Oliver P, Tibbetts S, et al.
J Virol
. 2023 Nov;
97(12):e0100823.
PMID: 37962378
The human gammaherpesviruses Epstein-Barr virus and Kaposi's sarcoma-associated herpesvirus are etiologic agents of numerous B cell lymphomas. A hallmark of gammaherpesvirus infection is their ability to establish lifelong latency in...
3.
Dar A, Kim D, Gordon S, Klinzing K, Rosen S, Guha I, et al.
Nat Commun
. 2023 Nov;
14(1):7098.
PMID: 37925424
During infection, virus-specific CD8 T cells undergo rapid bursts of proliferation and differentiate into effector cells that kill virus-infected cells and reduce viral load. This rapid clonal expansion can put...
4.
Kumar B, Field N, Kim D, Dar A, Chen Y, Suresh A, et al.
Nat Commun
. 2022 May;
13(1):2786.
PMID: 35589717
Antigen encounter directs CD4 T cells to differentiate into T helper or regulatory cells. This process focuses the immune response on the invading pathogen and limits tissue damage. Mechanisms that...
5.
Dar A, Sawada K, Dybas J, Moser E, Lewis E, Park E, et al.
PLoS Biol
. 2021 Feb;
19(2):e3001041.
PMID: 33524014
The capacity for T cells to become activated and clonally expand during pathogen invasion is pivotal for protective immunity. Our understanding of how T cell receptor (TCR) signaling prepares cells...
6.
Field N, Elbulok O, Dybas J, Moser E, Dar A, Spruce L, et al.
Eur J Immunol
. 2020 May;
50(10):1468-1483.
PMID: 32459862
To mount an antipathogen response, CD4 T cells must undergo rapid cell proliferation; however, poorly controlled expansion can result in diseases such as autoimmunity. One important regulator of T-cell activity...
7.
Field N, Moser E, Oliver P
J Leukoc Biol
. 2020 May;
108(1):353-362.
PMID: 32356405
The E3 ubiquitin ligase Itch has long been appreciated to be a critical suppressor of inflammation, first identified as a regulator of Th2 differentiation and lung inflammation. Recent studies have...
8.
Field N, OLeary C, Dybas J, Ding H, Oliver P
Methods Mol Biol
. 2020 Jan;
2111:239-256.
PMID: 31933212
Ubiquitination is a crucial component of many immune processes. While ubiquitin-mediated degradation is essential to T cell activation via T cell receptor signaling, the specific E3 ligases and substrates involved...
9.
Moser E, Roof J, Dybas J, Spruce L, Seeholzer S, Cancro M, et al.
J Exp Med
. 2019 Jul;
216(9):2170-2183.
PMID: 31311822
The E3 ubiquitin ligase Itch regulates antibody levels and prevents autoimmune disease in humans and mice, yet how Itch regulates B cell fate or function is unknown. We now show...
10.
Moser E, Oliver P
Cell Immunol
. 2019 May;
340:103916.
PMID: 31126634
Itch is a HECT type E3 ubiquitin ligase that is required to prevent the development of autoimmune disease in both mice and humans. Itch is expressed in most mammalian cell...