L J Striker
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Explore the profile of L J Striker including associated specialties, affiliations and a list of published articles.
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98
Citations
1400
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Recent Articles
1.
Zheng F, Zeng Y, Plati A, Elliot S, Berho M, Potier M, et al.
Kidney Int
. 2006 Jun;
70(3):507-14.
PMID: 16775596
The accumulation of advanced glycation end products (AGE) is a key factor in diabetic nephropathy (DN). Pyridoxamine inhibits AGE formation and protects against type I DN. Herein we tested: (1)...
2.
Elliot S, Zorn B, McLeod D, Moul J, Nyberg L, Striker L, et al.
Prostate Cancer Prostatic Dis
. 2003 Jun;
6(2):138-42.
PMID: 12806372
Benign prostatic hyperplasia (BPH) involves proliferation of smooth muscle cells and increased deposition of extracellular matrix (ECM). We recently found that pentosan polysulfate (PPS) has marked effects on growth and...
3.
Cornacchia F, Fornoni A, Plati A, Thomas A, Wang Y, Inverardi L, et al.
J Clin Invest
. 2001 Dec;
108(11):1649-56.
PMID: 11733560
We found that ROP Os/+ (Os/+) mice had diffuse glomerulosclerosis and glomerular hypertrophy and that their mesangial cells (the vascular smooth muscle cells of the glomerulus) displayed an apparent sclerosing...
4.
Schwedler S, Verbeke P, Bakala H, Weiss M, Vilar J, Depreux P, et al.
Diabetes Obes Metab
. 2001 Aug;
3(4):230-9.
PMID: 11520302
Aims: Advanced glycation end products (AGE), which form from the non-enzymatic reaction of proteins and sugars, have been implicated in the pathogenesis of diabetic nephropathy. Recently, a compound [N-phenacylthiazolium bromide...
5.
Doi S, Rasaiah S, Tack I, Mysore J, Kopchick J, Moore J, et al.
Am J Nephrol
. 2001 Aug;
21(4):331-9.
PMID: 11509807
A low-protein (LP) diet has been associated with amelioration of renal function in glomerulosclerosis (GS). However, the mechanisms involved are still unclear. We have used a mouse transgenic for bovine...
6.
Lambert G, Sakai N, Vaisman B, Neufeld E, Marteyn B, Chan C, et al.
J Biol Chem
. 2001 Mar;
276(18):15090-8.
PMID: 11278414
To evaluate the biochemical and molecular mechanisms leading to glomerulosclerosis and the variable development of atherosclerosis in patients with familial lecithin cholesterol acyl transferase (LCAT) deficiency, we generated LCAT knockout...
7.
Fukada J, Schena S, Tack I, Ruiz P, Kurimoto Y, Pang M, et al.
Transplant Proc
. 2001 Mar;
33(1-2):536-7.
PMID: 11266945
No abstract available.
8.
Fornoni A, Cornacchia F, HOWARD G, Roos B, Striker G, Striker L
Nephrol Dial Transplant
. 2001 Mar;
16(3):500-5.
PMID: 11239022
Background: Immunosuppressant therapy is thought to be a major contributor to post-transplant bone disease. Histological data and serum parameters suggest that Cyclosporin A (CsA) treatment causes osteopenia as a result...
9.
Fornoni A, Li H, Foschi A, Striker G, Striker L
Am J Pathol
. 2001 Jan;
158(1):275-80.
PMID: 11141501
Cyclosporin A (CsA) nephropathy is associated with altered expression of apoptosis regulatory genes such as Fas-ligand and Bcl-2 family members in the glomerular, tubulointerstitial, and vascular compartments. Both hepatocyte growth...
10.
Fornoni A, Lenz O, Tack I, Potier M, Elliot S, Striker L, et al.
Transplantation
. 2000 Sep;
70(4):587-93.
PMID: 10972214
Background: Chronic nephrotoxicity is an important adverse effect of cyclosporine A (CsA) therapy. Tubulo-interstitial lesions and arteriolopathy are common histologic findings. Glomerular lesions are also described, but they are of...