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L J Striker

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Articles 98
Citations 1400
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Recent Articles
11.
Elliot S, Striker L, Connor E, McQuinn W, Blagg C, Striker G
Clin Nephrol . 2000 Sep; 54(2):121-7. PMID: 10968687
Background: Vascular access failure is a major cause of morbidity, and increased costs in patients undergoing maintenance hemodialysis. Stenosis, the most common underlying cause of loss of patency in failed...
12.
Fukada J, Schena S, Tack I, Ruiz P, Kurimoto Y, Pang M, et al.
Circ Res . 2000 Jul; 87(1):66-72. PMID: 10884374
Although systemic administration of NO donors has been shown to attenuate the development of neointimal hyperplasia in the balloon injury model, this strategy has not been tested in a model...
13.
Doublier S, Seurin D, Fouqueray B, Verpont M, Callard P, Striker L, et al.
Kidney Int . 2000 Jun; 57(6):2299-307. PMID: 10844600
Background: The growth hormone (GH)/insulin-like growth factor (IGF) system is thought to participate in the glomerulosclerosis process. Because IGF-binding proteins (IGFBPs) modulate IGF actions and hence GH secretion, this study...
14.
Bellush L, Doublier S, Holland A, Striker L, Striker G, Kopchick J
Endocrinology . 1999 Dec; 141(1):163-8. PMID: 10614635
To further investigate the role of GH in diabetic nephropathy, experimental diabetes was induced with streptozotocin (STZ) in mice in which the GH receptor/binding protein gene was disrupted. Body weight,...
15.
Schwedler S, Bobadilla N, Striker L, VAAMONDE C, Herrera-Acosta J, Striker G
Transplantation . 1999 Dec; 68(10):1583-8. PMID: 10589959
Background: Long-term cyclosporine (CsA) treatment leads to a decreased glomerular filtration rate, hyalinosis of afferent arterioles, and striped cortical tubulo-interstitial fibrosis. We showed previously that pentosan polysulfate (SP54) prevented the...
16.
Schwedler S, Gilbert T, Moreau E, Striker L, Merlet-Benichou C, Striker G
Kidney Int . 1999 Nov; 56(5):1683-90. PMID: 10571776
Background: We have previously found that nephron number was not fixed, that is, there was a direct correlation between low birth weight and decreased nephron number in infants. In sclerosis-prone...
17.
Lupia E, Elliot S, Lenz O, Zheng F, Hattori M, Striker G, et al.
Diabetes . 1999 Jul; 48(8):1638-44. PMID: 10426384
Nonobese diabetic (NOD) mice develop glomerulosclerosis shortly after the onset of diabetes. We showed that mesangial cells (MCs) from diabetic mice exhibited a stable phenotypic switch, consisting of both increased...
18.
Esposito C, He C, Striker G, Zalups R, Striker L
Am J Pathol . 1999 Mar; 154(3):891-7. PMID: 10079267
Nephron reduction is an important factor in the development of glomerulosclerosis. In a study of the oligosyndactyly (Os) mutation that causes a congenital 50% reduction in nephron number, we previously...
19.
Elliot S, Striker L, Stetler-Stevenson W, Jacot T, Striker G
J Am Soc Nephrol . 1999 Jan; 10(1):62-8. PMID: 9890310
Glomerulosclerosis is characterized by extracellular matrix accumulation and is often associated with mesangial cell proliferation. Heparin-like molecules have been shown to decrease glomerulosclerosis in vivo, although their cellular site and...
20.
Lenz O, Zheng F, Vilar J, Doublier S, Lupia E, Schwedler S, et al.
Nephrol Dial Transplant . 1998 Dec; 13(12):3074-8. PMID: 9870468
Background: Glomerulosclerosis, the common terminal event in chronic glomerular diseases such as diabetic nephropathy or IgA nephropathy, leads to end-stage renal disease. The considerable variation in both the risk of...