Julie A Schneider
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Explore the profile of Julie A Schneider including associated specialties, affiliations and a list of published articles.
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Recent Articles
1.
Zammit A, Yu L, Poole V, Arfanakis K, Schneider J, Petyuk V, et al.
bioRxiv
. 2025 Mar;
PMID: 40060468
Importance: Psychological traits reflecting neuroticism, depressive symptoms, loneliness, and purpose in life are risk factors of AD dementia; however, the underlying biologic mechanisms of these associations remain largely unknown. Objective:...
2.
Vialle R, de Paiva Lopes K, Li Y, Ng B, Schneider J, Buchman A, et al.
Genome Med
. 2025 Mar;
17(1):20.
PMID: 40038788
Background: Alzheimer's disease (AD) is a complex neurodegenerative disorder with substantial genetic influence. While genome-wide association studies (GWAS) have identified numerous risk loci for late-onset AD (LOAD), the functional mechanisms...
3.
Bay S, Rodina A, Haut F, Roychowdhury T, Argyrousi E, Staniszewski A, et al.
Res Sq
. 2025 Feb;
PMID: 39989971
Alzheimer's disease (AD) progresses as a continuum, from preclinical stages to late-stage cognitive decline, yet the molecular mechanisms driving this progression remain poorly understood. Here, we provide a systems-level map...
4.
de Paiva Lopes K, Vialle R, Green G, Fujita M, Gaiteri C, Menon V, et al.
bioRxiv
. 2025 Feb;
PMID: 39975342
Alzheimer's Disease (AD) is marked by the accumulation of pathology, neuronal loss, and gliosis and frequently accompanied by cognitive decline. Understanding brain cell interactions is key to identifying new therapeutic...
5.
Cornelis M, Fazlollahi A, Bennett D, Schneider J, Ayton S
J Neurochem
. 2025 Feb;
169(2):e16309.
PMID: 39918201
Brain iron (Fe) dyshomeostasis is implicated in neurodegenerative diseases. Genome-wide association studies (GWAS) have identified plausible loci correlated with peripheral levels of Fe. Systemic organs and the brain share several...
6.
Agrawal S, Yu L, Leurgans S, Nag S, Barnes L, Bennett D, et al.
Alzheimers Dement
. 2025 Jan;
21(2):e14500.
PMID: 39888320
Introduction: This study investigates the inter-related roles of hippocampal neuronal loss (HNL), limbic-predominant age-related TAR-DNA binding protein of 43 kDa (TDP-43) encephalopathy neuropathologic changes (LATE-NC), and Alzheimer's disease neuropathologic changes...
7.
Durant A, Mukherjee S, Lee M, Choi S, Scollard P, Klinedinst B, et al.
medRxiv
. 2025 Jan;
PMID: 39830268
Importance: "SuperAgers" are oldest-old adults (ages 80+) whose memory performance resembles that of adults in their 50s to mid-60s. Factors underlying their exemplary memory are underexplored in large, racially diverse...
8.
Wolk D, Nelson P, Apostolova L, Arfanakis K, Boyle P, Carlsson C, et al.
Alzheimers Dement
. 2025 Jan;
21(1):e14202.
PMID: 39807681
Limbic predominant age-related TDP-43 encephalopathy neuropathologic change (LATE-NC) is highly prevalent in late life and a common co-pathology with Alzheimer's disease neuropathologic change (ADNC). LATE-NC is a slowly progressive, amnestic...
9.
Seto M, Clifton M, Gomez M, Coughlan G, Gifford K, Jefferson A, et al.
bioRxiv
. 2025 Jan;
PMID: 39803447
The biological mechanisms underlying women's increased Alzheimer's disease (AD) prevalence remain undefined. Previous case/control studies have identified sex-biased molecular pathways, but sex-specific relationships between gene expression and AD endophenotypes, particularly...
10.
Phillips J, Schneider J, Bennett D, Crane P, Risacher S, Saykin A, et al.
Pac Symp Biocomput
. 2024 Dec;
30():488-503.
PMID: 39670391
Alzheimer's disease (AD) is a polygenic disorder with a prolonged prodromal phase, complicating early diagnosis. Recent research indicates that increased astrocyte reactivity is associated with a higher risk of pathogenic...