David A Bennett
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Explore the profile of David A Bennett including associated specialties, affiliations and a list of published articles.
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1307
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Recent Articles
1.
Zammit A, Yu L, Poole V, Arfanakis K, Schneider J, Petyuk V, et al.
bioRxiv
. 2025 Mar;
PMID: 40060468
Importance: Psychological traits reflecting neuroticism, depressive symptoms, loneliness, and purpose in life are risk factors of AD dementia; however, the underlying biologic mechanisms of these associations remain largely unknown. Objective:...
2.
Wood Alexander M, Honer W, Saloner R, Galea L, Bennett D, Rabin J, et al.
Sci Adv
. 2025 Mar;
11(10):eadt0757.
PMID: 40043118
Menopause is a major biological transition that may influence women's late-life brain health. Earlier estrogen depletion-via earlier menopause-has been associated with increased risk for Alzheimer's disease (AD). Synaptic dysfunction also...
3.
Ding H, Ye Z, Paschalidis A, Bennett D, Au R, Lin H
Alzheimers Dement
. 2025 Mar;
21(3):e70055.
PMID: 40042504
Introduction: The progressive nature of Alzheimer's disease (AD) highlights the importance of predicting lifetime risk and updating assessments as new data emerge. This study aimed to develop a dynamic model...
4.
Boyle P, Wang T, Mottola G, Stewart C, Wilson R, Bennett D, et al.
Alzheimers Dement
. 2025 Mar;
21(3):e14544.
PMID: 40042433
Introduction: The association of scam susceptibility with the timing of Alzheimer's disease (AD) dementia onset is unknown. Methods: One thousand ninety-two older adults without dementia underwent assessments of scam susceptibility...
5.
Vialle R, de Paiva Lopes K, Li Y, Ng B, Schneider J, Buchman A, et al.
Genome Med
. 2025 Mar;
17(1):20.
PMID: 40038788
Background: Alzheimer's disease (AD) is a complex neurodegenerative disorder with substantial genetic influence. While genome-wide association studies (GWAS) have identified numerous risk loci for late-onset AD (LOAD), the functional mechanisms...
6.
Haghayegh S, Herzog R, Bennett D, Redline S, Yaffe K, Stone K, et al.
J Alzheimers Dis
. 2025 Mar;
:13872877251319742.
PMID: 40035493
BackgroundEarly identification of individuals at risk for cognitive impairment is crucial, as the preclinical phase offers an opportunity for interventions to slow disease progression and improve outcomes.ObjectiveWhile sleep electroencephalography (EEG)...
7.
Humphrey J, Brophy E, Kosoy R, Zeng B, Coccia E, Mattei D, et al.
Nat Genet
. 2025 Mar;
57(3):604-615.
PMID: 40033057
Microglia, the innate immune cells of the central nervous system, have been genetically implicated in multiple neurodegenerative diseases. Mapping the genetics of gene expression in human microglia has identified several...
8.
Bhattacharya A, Fon E, Dagher A, Iturria-Medina Y, Stratton J, Savignac C, et al.
bioRxiv
. 2025 Mar;
PMID: 40027681
Historically, Alzheimer's disease (AD) and Parkinson's disease (PD) have been investigated as two distinct disorders of the brain. However, a few similarities in neuropathology and clinical symptoms have been documented...
9.
Sin M, Dage J, Nho K, Dowling N, Seyfried N, Bennett D, et al.
J Clin Med
. 2025 Feb;
14(4).
PMID: 40004604
Anti-amyloid therapies (AATs) are increasingly being recognized as promising treatment options for Alzheimer's disease (AD). Amyloid-related imaging abnormalities (ARIAs), small areas of edema and microbleeds in the brain presenting as...
10.
Arnold M, Buyukozkan M, Doraiswamy P, Nho K, Wu T, Gudnason V, et al.
Nat Commun
. 2025 Feb;
16(1):1910.
PMID: 39994231
Impaired glucose uptake in the brain is an early presymptomatic manifestation of Alzheimer's disease (AD), with symptom-free periods of varying duration that likely reflect individual differences in metabolic resilience. We...