Jai-Hyun Kim
Overview
Explore the profile of Jai-Hyun Kim including associated specialties, affiliations and a list of published articles.
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Articles
14
Citations
277
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0
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Recent Articles
1.
Oh S, Kim W, Kim J, Younes M, El-Naggar A, Myers J, et al.
Clin Cancer Res
. 2019 Aug;
25(15):4861.
PMID: 31371311
No abstract available.
2.
Parmakhtiar B, Burger R, Kim J, Fruehauf J
Mol Cancer Res
. 2019 May;
17(8):1675-1686.
PMID: 31088908
Ovarian cancer growth under hypoxic conditions results in hypoxia-inducible factor-1α (HIF1α) stabilization. HIF1α is an adverse prognostic factor that may contribute to worse outcomes via its capacity to bind to...
3.
Suh Y, Kim J, Sung M, Boo H, Yun H, Lee S, et al.
Cancer Lett
. 2013 Jan;
332(1):102-9.
PMID: 23348700
In this study, we investigated the antitumor effects of deguelin in several human breast cancer cells in vitro and in vivo. Deguelin inhibited cell viability and the anchorage-dependent and anchorage-independent...
4.
Kim J, Peacock M, George S, Hughes C
Angiogenesis
. 2012 May;
15(3):497-509.
PMID: 22622516
ALK1 (ACVRL1) is a member of the TGFβ receptor family and is expressed predominantly by arterial endothelial cells (EC). Mutations in ACVRL1 are responsible for hereditary hemorrhagic telangiectasia type 2...
5.
Oh S, Kim W, Lee O, Kang J, Woo J, Kim J, et al.
Cancer Sci
. 2012 Apr;
103(7):1259-66.
PMID: 22494072
Angiogenesis, the process by which new blood vessels are recruited to existing ones, is essential for tumor development. Insulin-like growth factor (IGF) binding protein-3 (IGFBP-3), which modulates bioavailability of IGF,...
6.
Benzinou M, Clermont F, Letteboer T, Kim J, Espejel S, Harradine K, et al.
Nat Commun
. 2012 Jan;
3:616.
PMID: 22233626
Hereditary haemorrhagic telangiectasia (HHT) [corrected] is a vascular dysplasia syndrome caused by mutations in transforming growth factor-β/bone morphogenetic protein pathway genes, ENG and ACVRL1. HHT [corrected] shows considerable variation in...
7.
Kim J, Choi D, Lee O, Oh S, Lippman S, Lee H
Blood
. 2011 May;
118(9):2622-31.
PMID: 21551235
Most antiangiogenic therapies currently being evaluated in clinical trials target the vascular endothelial growth factor pathway; however, the tumor vasculature can acquire resistance to vascular endothelial growth factor-targeted therapy by...
8.
Baruscotti I, Barchiesi F, Jackson E, Imthurn B, Stiller R, Kim J, et al.
Hypertension
. 2010 Jul;
56(3):397-404.
PMID: 20644008
Endothelial progenitor cells (EPCs) repair damaged endothelium and promote capillary formation, processes involving receptor tyrosine kinases (RTKs) and heme oxygenase 1 (HO-1). Because estradiol augments vascular repair, we hypothesize that...
9.
Crampton S, Wu B, Park E, Kim J, Solomon C, Waterman M, et al.
PLoS One
. 2009 Nov;
4(11):e7841.
PMID: 19924227
Background: THE COMPLEXITY OF WNT SIGNALING LIKELY STEMS FROM TWO SOURCES: multiple pathways emanating from frizzled receptors in response to wnt binding, and modulation of those pathways and target gene...
10.
Kim J, Zhao Y, Pan X, He X, Gilbert H
J Biol Chem
. 2009 Feb;
284(16):10400-8.
PMID: 19233841
Pdi1p (protein-disulfide isomerase) is a folding assistant of the endoplasmic reticulum (ER) that catalyzes disulfide formation and the isomerization of incorrect disulfides. Its disulfide forming activity is its essential function...