David Barit
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Explore the profile of David Barit including associated specialties, affiliations and a list of published articles.
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15
Citations
748
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0
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Recent Articles
1.
Brook R, Wang J, Barit D, Ho P, Lim H
Res Pract Thromb Haemost
. 2024 Sep;
8(5):102520.
PMID: 39258175
Background: Chronic kidney disease (CKD) is associated with increased bleeding and thrombotic risks. Standard blood tests do not sufficiently quantify these risks. Global coagulation assays (GCAs) provide a more comprehensive...
2.
Gooley K, Williams P, Mack H, Zhu V, Langsford D, Pianta T, et al.
Ophthalmic Genet
. 2023 Aug;
44(5):417-422.
PMID: 37537573
Background: Pierson syndrome and X-linked Alport syndrome result from pathogenic variants in and , respectively, and both affect basement membranes in the kidney and the eye. This study describes the...
3.
Lim H, Lui B, Tacey M, Barit D, Patel S, Donnan G, et al.
Thromb Res
. 2023 May;
226:127-135.
PMID: 37150026
Background: Despite cardiovascular diseases and thrombosis being major causes of death in patients with chronic kidney disease (CKD), there remains no effective biomarker to predict thrombotic risk in this population....
4.
Harraka P, Wightman T, Akom S, Sandhu K, Colville D, Catran A, et al.
Sci Rep
. 2022 Nov;
12(1):18301.
PMID: 36316518
Drusen are retinal deposits comprising cell debris, immune material and complement that are characteristic of macular degeneration but also found in glomerulonephritis. This was a pilot cross-sectional study to determine...
5.
Ham Y, Nicklason E, Wightman T, Akom S, Sandhu K, Harraka P, et al.
Kidney Int Rep
. 2022 May;
7(4):848-856.
PMID: 35497809
Introduction: Complement has been implicated in systemic lupus erythematosus (SLE) pathogenesis on the basis of the associations with inherited complement defects and genome-wide association study risk alleles, glomerular deposits, reduced...
6.
Degen D, Kanellis J, Hughes P, Barit D, Ierino F
Nephrology (Carlton)
. 2016 May;
21(6):528.
PMID: 27188412
No abstract available.
7.
Thallas-Bonke V, Jha J, Gray S, Barit D, Haller H, Schmidt H, et al.
Physiol Rep
. 2014 Nov;
2(11).
PMID: 25367693
Current treatments for diabetic nephropathy (DN) only result in slowing its progression, thus highlighting a need to identify novel targets. Increased production of reactive oxygen species (ROS) is considered a...
8.
Jha J, Gray S, Barit D, Okabe J, El-Osta A, Namikoshi T, et al.
J Am Soc Nephrol
. 2014 Feb;
25(6):1237-54.
PMID: 24511132
Diabetic nephropathy may occur, in part, as a result of intrarenal oxidative stress. NADPH oxidases comprise the only known dedicated reactive oxygen species (ROS)-forming enzyme family. In the rodent kidney,...
9.
You Y, Okada S, Ly S, Jandeleit-Dahm K, Barit D, Namikoshi T, et al.
Am J Physiol Renal Physiol
. 2013 Feb;
304(7):F840-8.
PMID: 23389458
NADPH oxidase (Nox) isoforms have been implicated in contributing to diabetic microvascular complications, but the functional role of individual isoforms in diabetic kidney are unclear. Nox2, in particular, is highly...
10.
Kleinschnitz C, Grund H, Wingler K, Armitage M, Jones E, Mittal M, et al.
PLoS Biol
. 2010 Sep;
8(9).
PMID: 20877715
Ischemic stroke is the second leading cause of death worldwide. Only one moderately effective therapy exists, albeit with contraindications that exclude 90% of the patients. This medical need contrasts with...