Vicki Thallas-Bonke
Overview
Explore the profile of Vicki Thallas-Bonke including associated specialties, affiliations and a list of published articles.
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Articles
30
Citations
1585
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0
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Recent Articles
1.
Tan S, Lindblom R, Ziemann M, Laskowski A, Granata C, Snelson M, et al.
Nutrients
. 2021 Apr;
13(5).
PMID: 33922959
Diabetic kidney disease (DKD) remains the number one cause of end-stage renal disease in the western world. In experimental diabetes, mitochondrial dysfunction in the kidney precedes the development of DKD....
2.
Snelson M, Tan S, Clarke R, de Pasquale C, Thallas-Bonke V, Nguyen T, et al.
Sci Adv
. 2021 Apr;
7(14).
PMID: 33789895
Intake of processed foods has increased markedly over the past decades, coinciding with increased microvascular diseases such as chronic kidney disease (CKD) and diabetes. Here, we show in rodent models...
3.
Thallas-Bonke V, Tan S, Lindblom R, Snelson M, Granata C, Jha J, et al.
Nephrol Dial Transplant
. 2020 Dec;
36(6):988-997.
PMID: 33367789
Background: The nicotinamide adenine dinucleotide phosphate oxidase isoform 4 (Nox4) mediates reactive oxygen species (ROS) production and renal fibrosis in diabetic kidney disease (DKD) at the level of the podocyte....
4.
Lindblom R, Higgins G, Nguyen T, Arnstein M, Henstridge D, Granata C, et al.
Clin Sci (Lond)
. 2020 Jan;
134(2):239-259.
PMID: 31943002
Mitochondrial stress has been widely observed in diabetic kidney disease (DKD). Cyclophilin D (CypD) is a functional component of the mitochondrial permeability transition pore (mPTP) which allows the exchange of...
5.
Tan S, Ziemann M, Thallas-Bonke V, Snelson M, Kumar V, Laskowski A, et al.
Diabetes
. 2019 Oct;
69(1):83-98.
PMID: 31624141
The sequelae of diabetes include microvascular complications such as diabetic kidney disease (DKD), which involves glucose-mediated renal injury associated with a disruption in mitochondrial metabolic agility, inflammation, and fibrosis. We...
6.
Zhuang A, Yap F, McCarthy D, Leung C, Sourris K, Penfold S, et al.
Sci Rep
. 2019 Sep;
9(1):13664.
PMID: 31541173
The accumulation of advanced glycation end products (AGEs) have been implicated in the development and progression of diabetic kidney disease (DKD). There has been interest in investigating the potential of...
7.
Jha J, Banal C, Okabe J, Gray S, Hettige T, Chow B, et al.
Diabetes
. 2017 Jul;
66(10):2691-2703.
PMID: 28747378
NADPH oxidase-derived excessive production of reactive oxygen species (ROS) in the kidney plays a key role in mediating renal injury in diabetes. Pathological changes in diabetes include mesangial expansion and...
8.
Coughlan M, Nguyen T, Penfold S, Higgins G, Thallas-Bonke V, Tan S, et al.
Clin Sci (Lond)
. 2016 Feb;
130(9):711-20.
PMID: 26831938
Oxidative phosphorylation (OXPHOS) drives ATP production by mitochondria, which are dynamic organelles, constantly fusing and dividing to maintain kidney homoeostasis. In diabetic kidney disease (DKD), mitochondria appear dysfunctional, but the...
9.
Coughlan M, Higgins G, Nguyen T, Penfold S, Thallas-Bonke V, Tan S, et al.
Diabetes
. 2016 Jan;
65(4):1085-98.
PMID: 26822084
Apoptosis-inducing factor (AIF) is a mitochondrial flavoprotein with dual roles in redox signaling and programmed cell death. Deficiency in AIF is known to result in defective oxidative phosphorylation (OXPHOS), via...
10.
Jha J, Thallas-Bonke V, Banal C, Gray S, Chow B, Ramm G, et al.
Diabetologia
. 2015 Oct;
59(2):379-89.
PMID: 26508318
Aims/hypothesis: Changes in podocyte morphology and function are associated with albuminuria and progression of diabetic nephropathy. NADPH oxidase 4 (NOX4) is the main source of reactive oxygen species (ROS) in...