Chris Gaiteri
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Explore the profile of Chris Gaiteri including associated specialties, affiliations and a list of published articles.
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54
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Recent Articles
1.
Barnett E, Hess J, Hou J, Escott-Price V, Fennema-Notestine C, Kremen W, et al.
medRxiv
. 2025 Mar;
PMID: 40061341
Background: Genetic risk factors for psychiatric and neurodegenerative disorders are well documented. However, some individuals with high genetic risk remain unaffected, and the mechanisms underlying such resilience remain poorly understood....
2.
Vialle R, de Paiva Lopes K, Li Y, Ng B, Schneider J, Buchman A, et al.
Genome Med
. 2025 Mar;
17(1):20.
PMID: 40038788
Background: Alzheimer's disease (AD) is a complex neurodegenerative disorder with substantial genetic influence. While genome-wide association studies (GWAS) have identified numerous risk loci for late-onset AD (LOAD), the functional mechanisms...
3.
de Paiva Lopes K, Vialle R, Green G, Fujita M, Gaiteri C, Menon V, et al.
bioRxiv
. 2025 Feb;
PMID: 39975342
Alzheimer's Disease (AD) is marked by the accumulation of pathology, neuronal loss, and gliosis and frequently accompanied by cognitive decline. Understanding brain cell interactions is key to identifying new therapeutic...
4.
Hou J, Hess J, Zhang C, van Rooij J, Hearn G, Fan C, et al.
Am J Med Genet B Neuropsychiatr Genet
. 2024 Dec;
:e33019.
PMID: 39679839
The comprehensive genome-wide nature of transcriptome studies in Alzheimer's disease (AD) should provide a reliable description of disease molecular states. However, the genes and molecular systems nominated by transcriptomic studies...
5.
Ng B, Avey D, de Paiva Lopes K, Fujita M, Vialle R, Vyas H, et al.
bioRxiv
. 2024 Nov;
PMID: 39554066
Long non-coding RNAs (lncRNAs) are critical regulators of physiological and pathological processes, with their dysregulation increasingly implicated in aging and Alzheimer's disease (AD). Using spatial transcriptomics, we analyzed 78 postmortem...
6.
Tasaki S, Avey D, Kearns N, Iatrou A, Yu C, Tissera S, et al.
bioRxiv
. 2024 Nov;
PMID: 39484606
The gene signatures of Alzheimer's Disease (AD) brains reflect an output of a complex interplay of genetic, epigenetic, epi-transcriptomic, and post-transcriptional regulations. To identify the most significant factor that shapes...
7.
Ng B, Tasaki S, Greathouse K, Walker C, Zhang A, Covitz S, et al.
Nat Neurosci
. 2024 Nov;
27(11):2240-2252.
PMID: 39482360
Brain connectivity arises from interactions across biophysical scales, ranging from molecular to cellular to anatomical to network level. To date, there has been little progress toward integrated analysis across these...
8.
Avey D, Ng B, Vialle R, Kearns N, de Paiva Lopes K, Iatrou A, et al.
bioRxiv
. 2024 Sep;
PMID: 39314329
Amyloid-beta (Aβ) plaques and surrounding glial activation are prominent histopathological hallmarks of Alzheimer's Disease (AD). However, it is unclear how Aβ plaques interact with surrounding glial cells in the human...
9.
Binette A, Gaiteri C, Wennstrom M, Kumar A, Hristovska I, Spotorno N, et al.
Nat Neurosci
. 2024 Aug;
27(10):1880-1891.
PMID: 39187705
Proteomics can shed light on the dynamic and multifaceted alterations in neurodegenerative disorders like Alzheimer's disease (AD). Combining radioligands measuring β-amyloid (Aβ) plaques and tau tangles with cerebrospinal fluid proteomics,...
10.
Vialle R, de Paiva Lopes K, Li Y, Ng B, Schneider J, Buchman A, et al.
medRxiv
. 2024 Aug;
PMID: 39185527
Advances have led to a greater understanding of the genetics of Alzheimer's Disease (AD). However, the gap between the predicted and observed genetic heritability estimates when using single nucleotide polymorphisms...