Barry M Bradford
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Explore the profile of Barry M Bradford including associated specialties, affiliations and a list of published articles.
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28
Citations
596
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Recent Articles
1.
Stables J, Pal R, Bradford B, Carter-Cusack D, Taylor I, Pridans C, et al.
Neurobiol Dis
. 2024 Nov;
203:106743.
PMID: 39581554
Amino acid substitutions in the kinase domain of the human CSF1R protein are associated with autosomal dominant adult-onset leukoencephalopathy with axonal spheroids and pigmented glia (ALSP). To model the human...
2.
Bradford B, Walmsley-Rowe L, Reynolds J, Verity N, Mabbott N
Sci Rep
. 2024 Jun;
14(1):13749.
PMID: 38877012
Prion diseases are fatal, infectious, neurodegenerative disorders resulting from accumulation of misfolded cellular prion protein in the brain. Early pathological changes during CNS prion disease also include reactive astrocyte activation...
3.
Pal R, Bradford B, Mabbott N
Front Cell Neurosci
. 2022 Jul;
16:918883.
PMID: 35875357
Prion diseases such as Creutzfeldt-Jakob disease in humans, bovine spongiform encephalopathy in cattle, and scrapie in sheep, are infectious and chronic neurodegenerative diseases to which there are no cures. Infection...
4.
Bradford B, McGuire L, Hume D, Pridans C, Mabbott N
Glia
. 2022 Jul;
70(11):2169-2187.
PMID: 35852018
Prion diseases are transmissible, neurodegenerative disorders associated with misfolding of the prion protein. Previous studies show that reduction of microglia accelerates central nervous system (CNS) prion disease and increases the...
5.
Munro D, Bradford B, Mariani S, Hampton D, Vink C, Chandran S, et al.
Development
. 2020 Dec;
147(23).
PMID: 33323375
The central nervous system hosts parenchymal macrophages, known as microglia, and non-parenchymal macrophages, collectively termed border-associated macrophages (BAMs). Microglia, but not BAMs, were reported to be absent in mice lacking...
6.
Alterauge D, Bagnoli J, Dahlstrom F, Bradford B, Mabbott N, Buch T, et al.
Cell Rep
. 2020 Oct;
33(1):108232.
PMID: 33027650
T follicular helper (Tfh) cells are crucial for the establishment of germinal centers (GCs) and potent antibody responses. Nevertheless, the T cell-intrinsic factors that are required for the maintenance of...
7.
Mabbott N, Bradford B, Pal R, Young R, Donaldson D
Int J Mol Sci
. 2020 Oct;
21(19).
PMID: 33023255
Prion diseases are a unique group of infectious chronic neurodegenerative disorders to which there are no cures. Although prion infections do not stimulate adaptive immune responses in infected individuals, the...
8.
Alfituri O, Bradford B, Paxton E, Morrison L, Mabbott N
Front Immunol
. 2020 Jun;
11:1118.
PMID: 32582198
Infection of the mammalian host with African trypanosomes begins when the tsetse fly vector injects the parasites into the skin dermis during blood feeding. After injection into the skin, trypanosomes...
9.
Donaldson D, Bradford B, Else K, Mabbott N
Sci Rep
. 2020 Mar;
10(1):4554.
PMID: 32165661
Prion infections in the central nervous system (CNS) can cause extensive neurodegeneration. Systemic inflammation can affect the progression of some neurodegenerative disorders. Therefore, we used the gastrointestinal helminth pathogen Trichuris...
10.
Bradford B, Mabbott N
Sci Rep
. 2019 Dec;
9(1):19119.
PMID: 31836813
Prion diseases are a unique, infectious, neurodegenerative disorders that can affect animals and humans. Data from mouse transmissions show that efficient infection of the host after intravenous (IV) prion exposure...