B E Corkey
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Explore the profile of B E Corkey including associated specialties, affiliations and a list of published articles.
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Articles
83
Citations
2176
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Recent Articles
1.
Branstrom R, Leibiger I, Leibiger B, Klement G, Nilsson J, Arhem P, et al.
Diabetologia
. 2007 May;
50(8):1670-7.
PMID: 17522836
Aims/hypothesis: The pancreatic beta cell ATP-sensitive potassium (K(ATP)) channel, composed of the pore-forming alpha subunit Kir6.2, a member of the inward rectifier K+channel family, and the regulatory beta subunit sulfonylurea...
2.
Katzman S, Messerli M, Barry D, Grossman A, Harel T, Wikstrom J, et al.
Am J Physiol Endocrinol Metab
. 2004 Sep;
287(6):E1090-9.
PMID: 15339741
The cells within the intact islet of Langerhans function as a metabolic syncytium, secreting insulin in a coordinated and oscillatory manner in response to external fuel. With increased glucose, the...
3.
Branstrom R, Aspinwall C, Valimaki S, Ostensson C, Tibell A, Eckhard M, et al.
Diabetologia
. 2004 Jan;
47(2):277-83.
PMID: 14740158
Aims/hypothesis: The ATP-regulated potassium (KATP) channel in the pancreatic beta cell couples the metabolic state to electrical activity. The primary regulator of the KATP channel is generally accepted to be...
4.
Yaney G, Corkey B
Diabetologia
. 2003 Sep;
46(10):1297-312.
PMID: 13680127
Increases in glucose or fatty acids affect metabolism via changes in long-chain acyl-CoA formation and chronically elevated fatty acids increase total cellular CoA. Understanding the response of pancreatic beta cells...
5.
Deeney J, Kohler M, KUBIK K, Brown G, Schultz V, Tornheim K, et al.
J Biol Chem
. 2001 Aug;
276(40):36946-50.
PMID: 11481328
Insulin secretion from glucose-stimulated pancreatic beta-cells is oscillatory, and this is thought to result from oscillations in glucose metabolism. One of the primary metabolic stimulus-secretion coupling factors is the ATP/ADP...
6.
Faradji R, Havari E, Chen Q, Gray J, Tornheim K, Corkey B, et al.
J Biol Chem
. 2001 Jul;
276(39):36695-702.
PMID: 11443115
We have shown that intermediate lobe (IL) pituitary cells can be engineered to produce sufficient amounts of insulin (ins) to cure diabetes in nonobese diabetic mice but, unlike transplanted islets,...
7.
Korchak H, Corkey B, Yaney G, Kilpatrick L
Am J Physiol Cell Physiol
. 2001 Jul;
281(2):C514-23.
PMID: 11443050
In phagocytic cells, fMet-Leu-Phe triggers phosphoinositide remodeling, activation of protein kinase C (PKC), release of intracellular Ca(2+) and uptake of extracellular Ca(2+). Uptake of extracellular Ca(2+) can be triggered by...
8.
Caserta F, Tchkonia T, Civelek V, Prentki M, Brown N, McGarry J, et al.
Am J Physiol Endocrinol Metab
. 2001 Feb;
280(2):E238-47.
PMID: 11158926
Regional differences in free fatty acid (FFA) handling contribute to diseases associated with particular fat distributions. As cultured rat preadipocytes became differentiated, FFA transfer into preadipocytes increased and was more...
9.
Yaney G, Civelek V, Richard A, Dillon J, Deeney J, Hamilton J, et al.
Diabetes
. 2001 Jan;
50(1):56-62.
PMID: 11147795
Glucagon-like peptide 1 (GLP-1) is the most potent physiological incretin for insulin secretion from the pancreatic beta-cell, but its mechanism of action has not been established. It interacts with specific...
10.
Dillon J, Yaney G, Zhou Y, Voilley N, Bowen S, Chipkin S, et al.
Diabetes
. 2000 Dec;
49(12):2012-20.
PMID: 11118002
Administration of dehydroepiandrosterone (DHEA), or its sulfated form (DHEAS), controls hyperglycemia in diabetic rodents without directly altering insulin sensitivity. We show that DHEAS enhanced glucose-stimulated insulin secretion when administered in...