M Prentki
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Explore the profile of M Prentki including associated specialties, affiliations and a list of published articles.
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83
Citations
3619
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Recent Articles
1.
Poursharifi P, Schmitt C, Chenier I, Leung Y, Oppong A, Bai Y, et al.
Mol Metab
. 2023 Oct;
78:101822.
PMID: 37838014
Objective: Pro-inflammatory polarization of adipose tissue macrophages (ATMs) plays a critical role in the pathogenesis of obesity-associated chronic inflammation. However, little is known about the role of lipids in the...
2.
Rancoule C, Attane C, Gres S, Fournel A, Dusaulcy R, Bertrand C, et al.
Diabetologia
. 2013 Mar;
56(6):1394-402.
PMID: 23508306
Aims/hypothesis: Lysophosphatidic acid (LPA) is a lipid mediator produced by adipocytes that acts via specific G-protein-coupled receptors; its synthesis is modulated in obesity. We previously reported that reducing adipocyte LPA...
3.
Ferdaoussi M, Bergeron V, Zarrouki B, Kolic J, Cantley J, Fielitz J, et al.
Diabetologia
. 2012 Jul;
55(10):2682-2692.
PMID: 22820510
Aims/hypothesis: Activation of the G protein-coupled receptor (GPR)40 by long-chain fatty acids potentiates glucose-stimulated insulin secretion (GSIS) from pancreatic beta cells, and GPR40 agonists are in clinical development for type...
4.
Fontes G, Zarrouki B, Hagman D, Latour M, Semache M, Roskens V, et al.
Diabetologia
. 2010 Jul;
53(11):2369-79.
PMID: 20628728
Aims/hypothesis: Prolonged exposure of pancreatic beta cells to excessive levels of glucose and fatty acids, referred to as glucolipotoxicity, is postulated to contribute to impaired glucose homeostasis in patients with...
5.
Delghingaro-Augusto V, Nolan C, Gupta D, Jetton T, Latour M, Peshavaria M, et al.
Diabetologia
. 2009 Mar;
52(6):1122-32.
PMID: 19294363
Aims/hypothesis: The Zucker fatty (ZF) rat subjected to 60% pancreatectomy (Px) develops moderate diabetes by 3 weeks. We determined whether a progressive fall in beta cell mass and/or beta cell...
6.
Coletta D, Sriwijitkamol A, Wajcberg E, Tantiwong P, Li M, Prentki M, et al.
Diabetologia
. 2009 Jan;
52(4):723-32.
PMID: 19169664
Aims/hypothesis: The molecular mechanisms by which thiazolidinediones improve insulin sensitivity in type 2 diabetes are not fully understood. We hypothesised that pioglitazone would activate the adenosine 5'-monophosphate-activated protein kinase (AMPK)...
7.
Nolan C, Leahy J, Delghingaro-Augusto V, Moibi J, Soni K, Peyot M, et al.
Diabetologia
. 2006 Jul;
49(9):2120-30.
PMID: 16868750
Aims/hypothesis: The aim of this study was to determine the role of fatty acid signalling in islet beta cell compensation for insulin resistance in the Zucker fatty fa/fa (ZF) rat,...
8.
Buteau J, El-Assaad W, Rhodes C, Rosenberg L, Joly E, Prentki M
Diabetologia
. 2004 Apr;
47(5):806-15.
PMID: 15095038
Aims/hypothesis: We have provided evidence that glucagon-like peptide-1, a potential therapeutic agent in the treatment of diabetes, activates phosphatidylinositol-3 kinase/protein kinase B signalling in the pancreatic beta cell. Since this...
9.
Ruderman N, Park H, Kaushik V, Dean D, Constant S, Prentki M, et al.
Acta Physiol Scand
. 2003 Jul;
178(4):435-42.
PMID: 12864749
Unlabelled: An increasing body of evidence has revealed that activation of adenosine monophosphate (AMP)-activated protein kinase (AMPK)-activated protein kinase increases fatty acid oxidation by lowering the concentration of malonyl coenzyme...
10.
Mason T, Chan B, Goh T, Gupta N, Gamble J, Qing Shi Z, et al.
Atherosclerosis
. 2002 Mar;
161(2):345-52.
PMID: 11888517
Chronic intraperitoneal or subcutaneous insulin administration increases triglyceride secretion rate (TGSR) in normal rats. We wished to determine the effect of this treatment on TGSR and the hepatic lipogenic enzymes...