Vijayasaradhi Setaluri
Overview
Explore the profile of Vijayasaradhi Setaluri including associated specialties, affiliations and a list of published articles.
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70
Citations
3312
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Recent Articles
1.
Teertam S, Setaluri V, Ayuso J
JID Innov
. 2024 Nov;
5(1):100315.
PMID: 39525704
The skin plays a critical role in human physiology, acting both as a barrier to environmental insults and as a window to environmental stimuli. Disruption of this homeostasis leads to...
2.
Lin K, Hibbert J, Flynn C, Lemens J, Torbey M, Steinert N, et al.
EMBO Rep
. 2024 Aug;
25(9):3812-3841.
PMID: 39143258
Satellite cells are skeletal muscle stem cells that contribute to postnatal muscle growth, and they endow skeletal muscle with the ability to regenerate after a severe injury. Here we discover...
3.
Lin K, Hibbert J, Lemens J, Torbey M, Steinert N, Flejsierowicz P, et al.
bioRxiv
. 2024 Jan;
PMID: 38187693
Satellite cells are skeletal muscle stem cells that contribute to postnatal muscle growth, and they endow skeletal muscle with the ability to regenerate after a severe injury. Here we discovered...
4.
Setaluri V, Helfaer E
Pigment Cell Melanoma Res
. 2023 Oct;
36(6):454.
PMID: 37903734
No abstract available.
5.
Castro-Perez E, Singh M, Sadangi S, Mela-Sanchez C, Setaluri V
Pigment Cell Melanoma Res
. 2023 May;
36(5):330-347.
PMID: 37132530
Melanoma, a lethal malignancy that arises from melanocytes, exhibits a multiplicity of clinico-pathologically distinct subtypes in sun-exposed and non-sun-exposed areas. Melanocytes are derived from multipotent neural crest cells and are...
6.
Mikheil D, Prabhakar K, Ng T, Teertam S, Longley B, Newton M, et al.
Cancers (Basel)
. 2023 Jan;
15(2).
PMID: 36672468
Both oncogenic and tumor suppressor roles have been assigned to Notch signaling in melanoma. In clinical trials, Notch inhibitors proved to be ineffective for melanoma treatment. Notch signaling has also...
7.
Singh M, Altameemi S, Lares M, Newton M, Setaluri V
Sci Rep
. 2022 Aug;
12(1):14395.
PMID: 35999349
Melanoma cells exhibit phenotypic plasticity that allows transition from a proliferative and differentiated phenotype to a more invasive and undifferentiated or transdifferentiated phenotype often associated with drug resistance. The mechanisms...
8.
Krishnan A, Bhasker A, Singh M, Rodriguez C, Perez E, Altameemi S, et al.
Mol Cancer Res
. 2022 Jul;
20(10):1548-1560.
PMID: 35834616
Implications: This study establishes loss of dependency on EPAC-mTORC1 signaling as hallmark of primary melanoma evolution and targeting this escape mechanism is a promising strategy for metastatic melanoma.
9.
Sadangi S, Milosavljevic K, Castro-Perez E, Lares M, Singh M, Altameemi S, et al.
Cancers (Basel)
. 2022 Mar;
14(5).
PMID: 35267541
is the most common mutation driver in melanoma. This mutation is known to cause a brief burst of proliferation followed by growth arrest and senescence, which prevent an uncontrolled cell...
10.
Ayuso J, Sadangi S, Lares M, Rehman S, Humayun M, Denecke K, et al.
Lab Chip
. 2021 Feb;
21(6):1139-1149.
PMID: 33533390
Melanoma evolution is a complex process. The role epidermal keratinocytes and dermal fibroblasts play in this process and the mechanisms involved in tumor-stroma interactions remain poorly understood. Here, we used...