Thomas Langmann
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Explore the profile of Thomas Langmann including associated specialties, affiliations and a list of published articles.
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155
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5705
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Recent Articles
1.
Ciurus S, Elewa M, Palmer M, Wolf A, Hector M, Fuhrmann D, et al.
Cell Mol Life Sci
. 2025 Feb;
82(1):85.
PMID: 39985685
Chronic inflammatory diseases are a significant global burden and are associated with dysregulated resolution of inflammation. Therefore, promoting the process of resolution is a promising therapeutic approach. This study presents...
2.
Kasper M, Karlstetter M, Wildschutz L, Scholz R, Busch M, Bauer D, et al.
J Neuroinflammation
. 2025 Feb;
22(1):37.
PMID: 39930455
In this study the retinal transcriptome was investigated during the development of experimental autoimmune uveoretinitis (EAU) in mice. EAU was induced by immunizing B10.RIII mice with human interphotoreceptor retinoid binding...
3.
Behnke V, Wolf A, Hector M, Langmann T
Invest Ophthalmol Vis Sci
. 2025 Jan;
66(1):15.
PMID: 39775695
Purpose: In the aging retina, persistent activation of microglia is known to play a key role in retinal degenerative diseases like age-related macular degeneration (AMD). Furthermore, dysregulation of the alternative...
4.
Cuevas-Rios G, Abou Assale T, Wissfeld J, Bungartz A, Hofmann J, Langmann T, et al.
Glia
. 2024 Sep;
72(12):2295-2312.
PMID: 39228105
Sialylation plays an important role in self-recognition, as well as keeping the complement and innate immune systems in check. It is unclear whether the reduced sialylation seen during aging and...
5.
Hector M, Langmann T, Wolf A
Prog Retin Eye Res
. 2024 Mar;
100:101249.
PMID: 38430990
Translocator protein (18 kDa) (Tspo), formerly known as peripheral benzodiazepine receptor is a highly conserved transmembrane protein primarily located in the outer mitochondrial membrane. In the central nervous system (CNS),...
6.
Laudenberg N, Kinuthia U, Langmann T
Front Immunol
. 2024 Jan;
14:1345382.
PMID: 38288111
Reactive microglia are a hallmark of age-related retinal degenerative diseases including age-related macular degeneration (AMD). These cells are capable of secreting neurotoxic substances that may aggravate inflammation that leads to...
7.
Nuzhat N, Van Schil K, Liakopoulos S, Bauwens M, Duenas Rey A, Kaseberg S, et al.
J Clin Invest
. 2023 Mar;
133(8).
PMID: 36862503
Defects in primary or motile cilia result in a variety of human pathologies, and retinal degeneration is frequently associated with these so-called ciliopathies. We found that homozygosity for a truncating...
8.
Hata M, Andriessen E, Hata M, Diaz-Marin R, Fournier F, Crespo-Garcia S, et al.
Science
. 2023 Jan;
379(6627):45-62.
PMID: 36603072
Age-related macular degeneration is a prevalent neuroinflammatory condition and a major cause of blindness driven by genetic and environmental factors such as obesity. In diseases of aging, modifiable factors can...
9.
Tabel M, Wolf A, Szczepan M, Xu H, Jagle H, Moehle C, et al.
J Neuroinflammation
. 2022 Sep;
19(1):229.
PMID: 36115971
Background: Dysfunctional humoral and cellular innate immunity are key components in the development and progression of age-related macular degeneration (AMD). Specifically, chronically activated microglia and their disturbed regulatory system contribute...
10.
Hansen J, Bruckner M, Pietrowski M, Jikeli J, Plescher M, Beckert H, et al.
Mol Biol Cell
. 2022 Jun;
33(11):ar99.
PMID: 35731557
Microglia are the primary resident innate immune cells of the CNS. They possess branched, motile cell processes that are important for their cellular functions. To study the pathways that control...