T K Bammler
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Explore the profile of T K Bammler including associated specialties, affiliations and a list of published articles.
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15
Citations
265
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Recent Articles
1.
Anderson G, Peterson T, Farin F, Bammler T, Beyer R, Kantor E, et al.
Front Neurosci
. 2013 Apr;
7:21.
PMID: 23550224
Microarray-based transcriptional profiling was used to determine the effect of nicotinamide on gene expression in an experimental traumatic brain injury (TBI) model. Ingenuity Pathway Analysis (IPA) was used to evaluate...
2.
Stamper B, Mecham B, Park S, Wilkerson H, Farin F, Beyer R, et al.
Physiol Genomics
. 2012 Oct;
44(23):1154-63.
PMID: 23073384
The discovery of causal mechanisms associated with nonsyndromic craniosynostosis has proven to be a difficult task due to the complex nature of the disease. In this study, differential transcriptome correlation...
3.
Gross-Steinmeyer K, Stapleton P, Tracy J, Bammler T, Lehman T, Strom S, et al.
Xenobiotica
. 2005 Jul;
35(5):419-38.
PMID: 16012075
1. Previous studies reported that rat hepatocytes overlaid with extracellular matrix components (Matrigel) maintain the expression and responsiveness of drug-metabolizing enzymes. However, whether Matrigel provides similar advantages in human hepatocytes...
4.
Gross-Steinmeyer K, Stapleton P, Liu F, Tracy J, Bammler T, Quigley S, et al.
Xenobiotica
. 2005 Jan;
34(7):619-32.
PMID: 15672752
1. The naturally occurring compounds curcumin (CUR), 3,3'-diindolylmethane (DIM), isoxanthohumol (IXN), 8-prenylnaringenin (8PN), phenethyl isothiocyanate (PEITC) and sulforaphane (SFN) protect animals against chemically induced tumours. Putative chemoprotective mechanisms include modulated...
5.
Eaton D, Bammler T, Kelly E
Adv Exp Med Biol
. 2002 Jan;
500:559-76.
PMID: 11764998
It is now evident that most, if not all, of the remarkable species differences in susceptibility to AFB hepatocarcinogenesis is due in large part, if not exclusively, to differences in...
6.
Wang C, Bammler T, Guo Y, Kelly E, Eaton D
Toxicol Sci
. 2000 Jun;
56(1):26-36.
PMID: 10869451
Mice are resistant to the carcinogenic effects of the mycotoxin aflatoxin B(1) (AFB(1)) because they constitutively express an alpha-class glutathione S-transferase (mGSTA3-3) that has high (approximately 200,000 pmol/min/mg) activity toward...
7.
Bammler T, Slone D, Eaton D
Toxicol Sci
. 2000 Apr;
54(1):30-41.
PMID: 10746929
Following aflatoxin B1 (AFB) exposure, rats readily develop liver tumors. However, treatment of rats with a variety of compounds, including the synthetic dithiolthione oltipraz and the antioxidant ethoxyquin, protects these...
8.
9.
Van Ness K, McHugh T, Bammler T, Eaton D
Toxicol Appl Pharmacol
. 1998 Oct;
152(1):166-74.
PMID: 9772212
Mice constitutively express glutathione S-transferase mGSTA3-3 in liver. This isoform possesses uniquely high conjugating activity toward aflatoxin B1-8,9-epoxide (AFBO), thereby protecting mice from aflatoxin B1-induced hepatocarcinogenicity. In contrast, rats constitutively...
10.
Buetler T, Bammler T, Hayes J, Eaton D
Cancer Res
. 1996 May;
56(10):2306-13.
PMID: 8625305
Oltipraz (OPZ) is currently being considered for human use to protect against aflatoxin B1 (AFB)-induced hepatocarcinogenesis based on its proven protective effect in rats. The effectiveness of this treatment presumes...