Sunil G Rattan
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Explore the profile of Sunil G Rattan including associated specialties, affiliations and a list of published articles.
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16
Citations
400
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Recent Articles
1.
de Oliveira Camargo R, Abualanaz B, Rattan S, Filomeno K, Dixon I
Wound Repair Regen
. 2021 Jun;
29(4):667-677.
PMID: 34076932
Heart disease with attendant cardiac fibrosis kills more patients in developed countries than any other disease, including cancer. We highlight the recent literature on factors that activate and also deactivate...
2.
Landry N, Rattan S, Dixon I
Methods Mol Biol
. 2021 May;
2299:171-179.
PMID: 34028743
Two-dimensional cell culture is the primary method employed for proof-of-concept studies in most molecular biology labs. While immortalized cell lines are convenient and easy to maintain for extended periods in...
3.
Landry N, Rattan S, Filomeno K, Meier T, Meier S, Foran S, et al.
Basic Res Cardiol
. 2021 Apr;
116(1):25.
PMID: 33847835
We have previously shown that overexpression of SKI, an endogenous TGF-β repressor, deactivates the pro-fibrotic myofibroblast phenotype in the heart. We now show that SKI also functions independently of SMAD/TGF-β...
4.
An Improved Method of Maintaining Primary Murine Cardiac Fibroblasts in Two-Dimensional Cell Culture
Landry N, Rattan S, Dixon I
Sci Rep
. 2019 Sep;
9(1):12889.
PMID: 31501457
Primary cardiac fibroblasts are notoriously difficult to maintain for extended periods of time in cell culture, due to the plasticity of their phenotype and sensitivity to mechanical input. In order...
5.
Dixon I, Landry N, Rattan S
Adv Exp Med Biol
. 2019 May;
1132:35-41.
PMID: 31037622
Cardiac muscle (the myocardium) is a unique arrangement of atria and ventricles that are spatially and electrically separated by a fibrous border. The spirally-arranged myocytes in both left and right...
6.
Landry N, Kavosh M, Filomeno K, Rattan S, Czubryt M, Dixon I
Physiol Rep
. 2018 Nov;
6(22):e13897.
PMID: 30488595
Many etiologies of heart disease are characterized by expansion and remodeling of the cardiac extracellular matrix (ECM or matrix) which results in cardiac fibrosis. Cardiac fibrosis is mediated in cardiac...
7.
Jahan F, Landry N, Rattan S, Dixon I, Wigle J
Int J Mol Sci
. 2018 Oct;
19(10).
PMID: 30336567
Following cardiac injury, fibroblasts are activated and are termed as myofibroblasts, and these cells are key players in extracellular matrix (ECM) remodeling and fibrosis, itself a primary contributor to heart...
8.
Gupta S, Zeglinski M, Rattan S, Landry N, Ghavami S, Wigle J, et al.
Oncotarget
. 2016 Oct;
7(48):78516-78531.
PMID: 27705938
The incidence of heart failure with concomitant cardiac fibrosis is very high in developed countries. Fibroblast activation in heart is causal to cardiac fibrosis as they convert to hypersynthetic cardiac...
9.
Zeglinski M, Davies J, Ghavami S, Rattan S, Halayko A, Dixon I
Biochim Biophys Acta
. 2016 Apr;
1863(6 Pt A):1261-8.
PMID: 27039037
Inappropriate cardiac interstitial remodeling is mediated by activated phenoconverted myofibroblasts. The synthesis of matrix proteins by these cells is triggered by both chemical and mechanical stimuli. Ski is a repressor...
10.
Ghavami S, Cunnington R, Yeganeh B, Davies J, Rattan S, Bathe K, et al.
Biochim Biophys Acta
. 2012 Oct;
1823(12):2274-86.
PMID: 23026405
Trans fats are not a homogeneous group of molecules and less is known about the cellular effects of individual members of the group. Vaccenic acid (VA) and elaidic acid (EA)...