Shreyas Niphadkar
Overview
Explore the profile of Shreyas Niphadkar including associated specialties, affiliations and a list of published articles.
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5
Citations
7
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Recent Articles
1.
Vengayil V, Niphadkar S, Adhikary S, Varahan S, Laxman S
Elife
. 2024 Sep;
12.
PMID: 39324403
Many cells in high glucose repress mitochondrial respiration, as observed in the Crabtree and Warburg effects. Our understanding of biochemical constraints for mitochondrial activation is limited. Using a screen, we...
2.
Kumar R, Zhang F, Niphadkar S, Onu C, Vijjamarri A, Greenberg M, et al.
bioRxiv
. 2024 Sep;
PMID: 39257769
Degradation of many yeast mRNAs involves decapping by the Dcp1:Dcp2 complex. Previous studies on decapping activators Edc3 and Scd6 suggested their limited roles in mRNA decay. RNA-seq analysis of mutants...
3.
Racine L, Parmentier R, Niphadkar S, Chhun J, Martignoles J, Delhommeau F, et al.
Life Sci Alliance
. 2024 May;
7(8).
PMID: 38802246
A continuous supply of energy is an essential prerequisite for survival and represents the highest priority for the cell. We hypothesize that cell differentiation is a process of optimization of...
4.
Niphadkar S, Karinje L, Laxman S
PLoS Genet
. 2024 Mar;
20(3):e1011202.
PMID: 38452140
To sustain growth in changing nutrient conditions, cells reorganize outputs of metabolic networks and appropriately reallocate resources. Signaling by reversible protein phosphorylation can control such metabolic adaptations. In contrast to...
5.
Shee S, Veetil R, Mohanraj K, Das M, Malhotra N, Bandopadhyay D, et al.
Elife
. 2023 Aug;
12.
PMID: 37642294
() is evolutionarily equipped to resist exogenous reactive oxygen species (ROS) but shows vulnerability to an increase in endogenous ROS (eROS). Since eROS is an unavoidable consequence of aerobic metabolism,...