Robert S Weinstein
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Explore the profile of Robert S Weinstein including associated specialties, affiliations and a list of published articles.
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57
Citations
6342
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Recent Articles
1.
Whyte M, Weinstein R, Phillips P, McAlister W, Ramakrishnaiah R, Schaefer G, et al.
Bone
. 2024 Jul;
188:117218.
PMID: 39084544
Craniotubular dysplasia, Ikegawa type (OMIM #619727) denotes the autosomal recessive skeletal disease identified in 2021 featuring blindness acquired in childhood. Five young members of four Indian families harbored a homozygous...
2.
Zhu M, Zeiss C, Hamrick M, Weinstein R, Sun B, Brotto M, et al.
Bone
. 2024 Mar;
184:117086.
PMID: 38552893
Purpose: Mitofusin 2 (Mfn2) is one of two mitofusins involved in regulating mitochondrial size, shape and function, including mitophagy, an important cellular mechanism to limit oxidative stress. Reduced expression of...
3.
Whyte M, Aronson J, McAlister W, Weinstein R, Wenkert D, Clements K, et al.
Bone
. 2020 Dec;
145:115835.
PMID: 33360005
In 2003, we briefly reported the remarkable osteopathy of a 12-year-old boy who at age two months began fracturing his limbs with subsequent hyperplastic callus formation and expansion and fusion...
4.
Menon L, Weinstein R
Bone
. 2019 Nov;
131:115137.
PMID: 31756522
Autosomal dominant hypophosphatemic rickets (ADHR) is remarkable among the hypophosphatemic rickets syndromes for its variable age of presentation and periods of quiescence during which serum phosphate and fibroblast growth factor...
5.
Ambrogini E, Que X, Wang S, Yamaguchi F, Weinstein R, Tsimikas S, et al.
Nat Commun
. 2018 Jun;
9(1):2193.
PMID: 29875355
Atherosclerosis and osteoporosis are epidemiologically linked and oxidation specific epitopes (OSEs), such as phosphocholine (PC) of oxidized phospholipids (PC-OxPL) and malondialdehyde (MDA), are pathogenic in both. The proatherogenic effects of...
6.
Weinstein R, Hogan E, Borrelli M, Liachenko S, OBrien C, Manolagas S
Endocrinology
. 2017 Sep;
158(11):3817-3831.
PMID: 28938402
In search of the sequence of pathogenic events leading to glucocorticoid-induced osteonecrosis, we determined the molecular, biomechanical, cellular, and vascular changes in the femur of C57BL/6 mice receiving prednisolone for...
7.
Piemontese M, Almeida M, Robling A, Kim H, Xiong J, Thostenson J, et al.
JCI Insight
. 2017 Sep;
2(17).
PMID: 28878136
Decreased cortical thickness and increased cortical porosity are the key anatomic changes responsible for osteoporotic fractures in elderly women and men. The cellular basis of these changes is unbalanced endosteal...
8.
Liu Y, Almeida M, Weinstein R, OBrien C, Manolagas S, Jilka R
Am J Physiol Endocrinol Metab
. 2016 Mar;
310(9):E762-73.
PMID: 26956187
ApoE-null (ApoE-KO) mice fed a high-fat diet (HFD) develop atherosclerosis, due in part to activation of vascular inflammation by oxidized low-density lipoprotein. Since bone loss also occurs in these mice,...
9.
Zhu M, Sun B, Saar K, Simpson C, Troiano N, Dallas S, et al.
J Bone Miner Res
. 2015 Oct;
31(4):864-73.
PMID: 26496249
Rac1 and Rac2 are thought to have important roles in osteoclasts. Therefore, mice with deletion of both Rac1 and Rac2 in mature osteoclasts (DKO) were generated by crossing Rac1(flox/flox) mice...
10.
Ucer S, Iyer S, Bartell S, Martin-Millan M, Han L, Kim H, et al.
J Bone Miner Res
. 2015 Feb;
30(7):1138-49.
PMID: 25704845
In men, androgens are critical for the acquisition and maintenance of bone mass in both the cortical and cancellous bone compartment. Male mice with targeted deletion of the androgen receptor...