Robert S Sloviter
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Explore the profile of Robert S Sloviter including associated specialties, affiliations and a list of published articles.
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29
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1568
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Recent Articles
1.
Bullinger K, Dhakar M, Pearson A, Bumanglag A, Guven E, Verma R, et al.
J Neurol
. 2025 Jan;
272(2):128.
PMID: 39812831
Objectives: The ability to differentiate epileptic- and non-epileptic events is challenging due to a lack of reliable molecular seizure biomarker that provide a retrospective diagnosis. Here, we use next generation...
2.
Chun E, Bumanglag A, Burke S, Sloviter R
Epilepsia
. 2019 Apr;
60(5):e52-e57.
PMID: 30963545
Cryptogenic temporal lobe epilepsy develops in the absence of identified brain injuries, infections, or structural malformations, and in these cases, an unidentified pre-existing abnormality may initiate febrile seizures, hippocampal sclerosis,...
3.
Bumanglag A, Sloviter R
Epilepsia
. 2018 Oct;
59(11):2019-2034.
PMID: 30338519
Objective: To determine when spontaneous granule cell epileptiform discharges first occur after hippocampal injury, and to identify the postinjury "latent" period as either a "silent" gestational state of epileptogenesis or...
4.
Klein P, Dingledine R, Aronica E, Bernard C, Blumcke I, Boison D, et al.
Epilepsia
. 2017 Dec;
59(1):37-66.
PMID: 29247482
The most common forms of acquired epilepsies arise following acute brain insults such as traumatic brain injury, stroke, or central nervous system infections. Treatment is effective for only 60%-70% of...
5.
Carpenter-Hyland E, Bichler E, Smith M, Sloviter R, Benveniste M
Physiol Rep
. 2017 Nov;
5(21).
PMID: 29138358
Hippocampal neuron plasticity is strongly associated with learning, memory, and cognition. In addition to modification of synaptic function and connectivity, the capacity of hippocampal neurons to undergo plasticity involves the...
6.
Sloviter R
Curr Opin Pharmacol
. 2017 Aug;
35:105-110.
PMID: 28781107
Pharmacological treatment to prevent brain injury-induced temporal lobe epileptogenesis has been generally unsuccessful, raising the issues of exactly when the conversion process to an epileptic brain state occurs and reaches...
7.
Dingledine R, Coulter D, Fritsch B, Gorter J, Lelutiu N, McNamara J, et al.
Sci Data
. 2017 May;
4:170061.
PMID: 28485718
Global expression profiling of neurologic or psychiatric disorders has been confounded by variability among laboratories, animal models, tissues sampled, and experimental platforms, with the result being that few genes demonstrate...
8.
Fares R, Belmeguenai A, Sanchez P, Kouchi H, Bodennec J, Morales A, et al.
PLoS One
. 2013 Jan;
8(1):e53888.
PMID: 23342033
Environmental enrichment of laboratory animals influences brain plasticity, stimulates neurogenesis, increases neurotrophic factor expression, and protects against the effects of brain insult. However, these positive effects are not constantly observed,...
9.
Sloviter R, Lomo T
Front Neural Circuits
. 2012 Dec;
6:102.
PMID: 23233836
Andersen et al. (1971) proposed that excitatory activity in the entorhinal cortex propagates topographically to the dentate gyrus, and on through a "trisynaptic circuit" lying within transverse hippocampal "slices" or...
10.
Sloviter R, Bumanglag A
Neuropharmacology
. 2012 Feb;
69:3-15.
PMID: 22342985
The "latent period" between brain injury and clinical epilepsy is widely regarded to be a seizure-free, pre-epileptic state during which a time-consuming cascade of molecular events and structural changes gradually...