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Argyle V Bumanglag

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Recent Articles
1.
Smith S, Garcia E, Davidson C, Thompson J, Lovett S, Ferekides N, et al.
Behav Brain Res . 2022 Sep; 437:114106. PMID: 36089100
Approximately 60-70 million people suffer from traumatic brain injury (TBI) each year. Animal models continue to be paramount in understanding mechanisms of cellular dysfunction and testing new treatments for TBI....
2.
Pais G, Chang J, Barreto E, Stitt G, Downes K, Alshaer M, et al.
Clin Pharmacokinet . 2022 Jun; 61(7):929-953. PMID: 35764774
Cefepime is a broad-spectrum fourth-generation cephalosporin with activity against Gram-positive and Gram-negative pathogens. It is generally administered as an infusion over 30-60 min or as a prolonged infusion with infusion...
3.
Altidor L, Bruner M, Deslauriers J, Garman T, Ramirez S, Dirr E, et al.
Neurobiol Learn Mem . 2021 Jul; 184:107498. PMID: 34332068
Cognitive flexibility is a prefrontal cortex-dependent neurocognitive process that enables behavioral adaptation in response to changes in environmental contingencies. Electrical vagus nerve stimulation (VNS) enhances several forms of learning and...
4.
Chun E, Bumanglag A, Burke S, Sloviter R
Epilepsia . 2019 Apr; 60(5):e52-e57. PMID: 30963545
Cryptogenic temporal lobe epilepsy develops in the absence of identified brain injuries, infections, or structural malformations, and in these cases, an unidentified pre-existing abnormality may initiate febrile seizures, hippocampal sclerosis,...
5.
Bumanglag A, Sloviter R
Epilepsia . 2018 Oct; 59(11):2019-2034. PMID: 30338519
Objective: To determine when spontaneous granule cell epileptiform discharges first occur after hippocampal injury, and to identify the postinjury "latent" period as either a "silent" gestational state of epileptogenesis or...
6.
Eid T, Behar K, Dhaher R, Bumanglag A, Lee T
Neurochem Res . 2012 Apr; 37(11):2339-50. PMID: 22488332
Glutamine synthetase (GS, E.C. 6.3.1.2) is a ubiquitous and highly compartmentalized enzyme that is critically involved in several metabolic pathways in the brain, including the glutamine-glutamate-GABA cycle and detoxification of...
7.
Sloviter R, Bumanglag A
Neuropharmacology . 2012 Feb; 69:3-15. PMID: 22342985
The "latent period" between brain injury and clinical epilepsy is widely regarded to be a seizure-free, pre-epileptic state during which a time-consuming cascade of molecular events and structural changes gradually...
8.
Norwood B, Bumanglag A, Osculati F, Sbarbati A, Marzola P, Nicolato E, et al.
J Comp Neurol . 2010 Jun; 518(16):3381-407. PMID: 20575073
In refractory temporal lobe epilepsy, seizures often arise from a shrunken hippocampus exhibiting a pattern of selective neuron loss called "classic hippocampal sclerosis." No single experimental injury has reproduced this...
9.
Bumanglag A, Sloviter R
J Comp Neurol . 2008 Aug; 510(6):561-80. PMID: 18697194
Hippocampal epileptogenesis is hypothesized to involve secondary mechanisms triggered by initial brain injury. Chemoconvulsant-induced status epilepticus has been used to identify secondary epileptogenic mechanisms under the assumption that a seizure-free,...
10.
Sloviter R, Zappone C, Bumanglag A, Norwood B, Kudrimoti H
Epilepsia . 2008 Mar; 48 Suppl 8:6-10. PMID: 18329985
No abstract available.