Ranjan Dutta
Overview
Explore the profile of Ranjan Dutta including associated specialties, affiliations and a list of published articles.
Author names and details appear as published. Due to indexing inconsistencies, multiple individuals may share a name, and a single author may have variations. MedLuna displays this data as publicly available, without modification or verification
Snapshot
Snapshot
Articles
102
Citations
3907
Followers
0
Related Specialties
Top 10 Co-Authors
Top 10 Co-Authors
Published In
Published In
Affiliations
Affiliations
Soon will be listed here.
Recent Articles
1.
Roy A, Dutta R, Halder D, Mandal K, Kundu S, Hossain M, et al.
Org Biomol Chem
. 2024 Dec;
23(5):1209-1214.
PMID: 39711100
-(tetra-aryl) picket calix[4]pyrrole 1 featuring -fluorophenyl groups at all four -positions in a -configuration has been synthesized and characterized unambiguously using single-crystal X-ray diffraction analysis. This pre-organized system possesses a...
2.
Yu M, Xu J, Dutta R, Trapp B, Pieper A, Cheng F
NPJ Syst Biol Appl
. 2024 Nov;
10(1):128.
PMID: 39500920
Amyotrophic Lateral Sclerosis (ALS) is a devastating, immensely complex neurodegenerative disease by lack of effective treatments. We developed a network medicine methodology via integrating human brain multi-omics data to prioritize...
3.
Poddar D, Sharma N, Ogino T, Qi X, Kessler P, Mendries H, et al.
mBio
. 2024 Jun;
15(7):e0056824.
PMID: 38888342
The interferon (IFN) system protects mammals from diseases caused by virus infections. IFN synthesis is induced by pattern recognition receptor signaling pathways activated by virus infection. IFN is secreted from...
4.
Khan D, Ramachandiran I, Vasu K, China A, Khan K, Cumbo F, et al.
Nat Commun
. 2024 May;
15(1):4284.
PMID: 38769304
Hypomyelinating leukodystrophy (HLD) is an autosomal recessive disorder characterized by defective central nervous system myelination. Exome sequencing of two siblings with severe cognitive and motor impairment and progressive hypomyelination characteristic...
5.
Ravichandar R, Gadelkarim F, Muthaiah R, Glynos N, Murlanova K, Rai N, et al.
J Neurosci
. 2024 May;
44(28.
PMID: 38749703
Dysregulation of oligodendrocyte progenitor cell (OPC) recruitment and oligodendrocyte differentiation contribute to failure of remyelination in human demyelinating diseases such as multiple sclerosis (MS). Deletion of muscarinic receptor enhances OPC...
6.
Liu X, Xin D, Zhong X, Zhao C, Li Z, Zhang L, et al.
Cell
. 2024 May;
187(10):2465-2484.e22.
PMID: 38701782
Remyelination failure in diseases like multiple sclerosis (MS) was thought to involve suppressed maturation of oligodendrocyte precursors; however, oligodendrocytes are present in MS lesions yet lack myelin production. We found...
7.
Yu M, Xu J, Dutta R, Trapp B, Pieper A, Cheng F
bioRxiv
. 2024 Apr;
PMID: 38585774
Amyotrophic Lateral Sclerosis (ALS) is a devastating, immensely complex neurodegenerative disease by lack of effective treatments. To date, the challenge to establishing effective treatment for ALS remains formidable, partly due...
8.
McCallum S, Suresh K, Islam T, Saustad A, Shelest O, Patil A, et al.
bioRxiv
. 2024 Apr;
PMID: 38558977
Spared regions of the damaged central nervous system undergo dynamic remodeling and exhibit a remarkable potential for therapeutic exploitation. Here, lesion-remote astrocytes (LRAs), which interact with viable neurons, glia and...
9.
Chomyk A, Kucinski R, Kim J, Christie E, Cyncynatus K, Gossman Z, et al.
Ann Neurol
. 2024 Feb;
95(5):907-916.
PMID: 38345145
Objective: Microglia/macrophages line the border of demyelinated lesions in both cerebral white matter and the cortex in the brains of multiple sclerosis patients. Microglia/macrophages associated with chronic white matter lesions...
10.
Marziali L, Hwang Y, Palmisano M, Cuenda A, Sim F, Gonzalez A, et al.
Brain
. 2023 Dec;
147(5):1871-1886.
PMID: 38128553
Multiple sclerosis is a chronic inflammatory disease in which disability results from the disruption of myelin and axons. During the initial stages of the disease, injured myelin is replaced by...