Preeti J Khandelwal
Overview
Explore the profile of Preeti J Khandelwal including associated specialties, affiliations and a list of published articles.
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11
Citations
383
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Recent Articles
1.
Khandelwal P, Dumanis S, Herman A, Rebeck G, Moussa C
Mol Cell Neurosci
. 2023 Mar;
125:103844.
PMID: 36966062
No abstract available.
2.
Herman A, Khandelwal P, Rebeck G, Moussa C
Exp Neurol
. 2012 Mar;
235(1):297-305.
PMID: 22402344
The transactivation DNA-binding protein (TDP-43) pathology is associated with fronto-temporal lobar dementia (FTLD) with ubiquitinated inclusions and some cases of Alzheimer's disease (AD). Proteolytic fragments of β-amyloid precursor protein (βAPP)...
3.
Distefano G, Gangemi A, Khandelwal P, Saunders A, Marenda D
Dev Dyn
. 2012 Jan;
241(3):553-62.
PMID: 22275119
Background: Proper neurogenesis in the developing Drosophila retina requires the regulated expression of the basic helix-loop-helix (bHLH) proneural transcription factors Atonal (Ato) and Daughterless (Da). Factors that control the timing...
4.
Khandelwal P, Dumanis S, Herman A, Rebeck G, Moussa C
Mol Cell Neurosci
. 2011 Sep;
49(1):44-53.
PMID: 21945393
This article has been retracted: please see Elsevier Policy on Article Withdrawal (http://www.elsevier.com/locate/withdrawalpolicy).
This article has been retracted at the request of the Editor-in-Chief.
Concerns about the article were raised...
5.
Khandelwal P, Herman A, Moussa C
J Neuroimmunol
. 2011 Aug;
238(1-2):1-11.
PMID: 21820744
Inflammation is secondary to protein accumulation in neurodegenerative diseases, including Alzheimer's, Parkinson's and Amyotrophic Lateral Sclerosis. Emerging evidence indicate sustained inflammatory responses, involving microglia and astrocytes in animal models of...
6.
Khandelwal P, Moussa C
Front Psychiatry
. 2011 Mar;
1:15.
PMID: 21423426
The most prominent changes in neurodegenerative diseases are protein accumulation and inclusion formation. Several neurodegenerative diseases, including Alzheimer's, the Synucleinopathies and Tauopathies share several overlapping clinical symptoms manifest in Parkinsonism,...
7.
Khandelwal P, Herman A, Hoe H, Rebeck G, Moussa C
Hum Mol Genet
. 2011 Mar;
20(11):2091-102.
PMID: 21378096
Intraneuronal amyloid-β (Aβ) may contribute to extracellular plaque deposition, the characteristic pathology of Alzheimer's disease (AD). The E3-ubiquitin ligase parkin ubiquitinates intracellular proteins and induces mitophagy. We previously demonstrated that...
8.
Herman A, Khandelwal P, Stanczyk B, Rebeck G, Moussa C
Brain Res
. 2011 Mar;
1386:191-9.
PMID: 21376022
Amyotrophic lateral sclerosis (ALS) is a neurodegenerative disease associated with loss of motor neurons in the brain and spinal cord. ALS is occasionally diagnosed with frontotemporal lobar dementia with ubiquitin-positive...
9.
Khandelwal P, Dumanis S, Feng L, Maguire-Zeiss K, Rebeck G, Lashuel H, et al.
Mol Neurodegener
. 2010 Nov;
5:47.
PMID: 21050448
Background: α-Synuclein aggregates in Lewy bodies and plays a central role in the pathogenesis of a group of neurodegenerative disorders, known as "Synucleinopathies", including Parkinson's disease. Parkin mutations result in...
10.
Jain S, Zhang X, Khandelwal P, Saunders A, Cummings B, Oelkers P
J Lipid Res
. 2009 Apr;
50(8):1563-70.
PMID: 19351971
Esterifying lysophospholipids may serve a variety of functions, including phospholipid remodeling and limiting the abundance of bioactive lipids. Recently, a yeast enzyme, Lpt1p, that esterifies an array of lysophospholipids was...