Paresh Prajapati
Overview
Explore the profile of Paresh Prajapati including associated specialties, affiliations and a list of published articles.
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Articles
29
Citations
509
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Recent Articles
1.
Niedowicz D, Wang W, Prajapati P, Zhong Y, Fister S, Rogers C, et al.
Brain Pathol
. 2024 Oct;
:e13312.
PMID: 39438022
Mutations in the human granulin (GRN) gene are associated with multiple diseases, including dementia disorders such as frontotemporal dementia (FTD) and limbic-predominant age-related TDP-43 encephalopathy (LATE). We studied a Grn...
2.
Kalimon O, Vekaria H, Prajapati P, Short S, Hubbard W, Sullivan P
Life (Basel)
. 2024 Aug;
14(8).
PMID: 39202703
Current literature finds females have improved outcomes over their male counterparts after severe traumatic brain injury (TBI), while the opposite seems to be true for mild TBI. This begs the...
3.
Vekaria H, Kalimon O, Prajapati P, Velmurugan G, Sullivan P
Front Mol Biosci
. 2024 Jul;
11:1378536.
PMID: 38983247
Mitochondrial function analysis is a well-established method used in preclinical and clinical investigations to assess pathophysiological changes in various disease states, including traumatic brain injury (TBI). Although there are multiple...
4.
Geleta U, Prajapati P, Bachstetter A, Nelson P, Wang W
Int J Mol Sci
. 2024 Mar;
25(5).
PMID: 38473893
Neurological diseases and neurotrauma manifest significant sex differences in prevalence, progression, outcome, and therapeutic responses. Genetic predisposition, sex hormones, inflammation, and environmental exposures are among many physiological and pathological factors...
5.
Hubbard W, Vekaria H, Velmurugan G, Kalimon O, Prajapati P, Brown E, et al.
J Neurotrauma
. 2023 Jul;
40(21-22):2396-2409.
PMID: 37476976
Mild traumatic brain injury (mTBI) results in impairment of brain metabolism, which is propagated by mitochondrial dysfunction in the brain. Mitochondrial dysfunction has been identified as a pathobiological therapeutic target...
6.
Velmurugan G, Hubbard W, Prajapati P, Vekaria H, Patel S, Rabchevsky A, et al.
Cells
. 2023 Jul;
12(10).
PMID: 37408279
The brain undergoes oxidative stress and mitochondrial dysfunction following physiological insults such as Traumatic brain injury (TBI), ischemia-reperfusion, and stroke. Pharmacotherapeutics targeting mitochondria (mitoceuticals) against oxidative stress include antioxidants, mild...
7.
Prajapati P, Wang W, Pesina S, Geleta U, Springer J
Cell Mol Neurobiol
. 2021 Nov;
43(1):423-429.
PMID: 34761332
Sex is a key biological variable in traumatic brain injury (TBI) and plays a significant role in neuroinflammatory responses. However, the molecular mechanisms contributing to this sexually dimorphic neuroinflammatory response...
8.
Gohel D, Sripada L, Prajapati P, Currim F, Roy M, Singh K, et al.
Free Radic Biol Med
. 2021 Jan;
165:100-110.
PMID: 33497798
Fragile X-associated tremor/ataxia syndrome (FXTAS) is a progressive neurodegenerative disorder caused by an expansion of 55 to 200 CGG repeats located within 5'UTR of FMR1.These CGG repeats are transcribed into...
9.
Wang W, Prajapati P, Vekaria H, Spry M, Cloud A, Sullivan P, et al.
Neural Regen Res
. 2020 Sep;
16(3):514-522.
PMID: 32985480
MicroRNAs (miRNAs) are small non-coding RNA molecules that regulate post-transcriptional gene expression and contribute to all aspects of cellular function. We previously reported that the activities of several mitochondria-enriched miRNAs...
10.
Prajapati P, Gohel D, Shinde A, Roy M, Singh K, Singh R
Cell Signal
. 2020 Sep;
76:109777.
PMID: 32918979
Emerging evidence suggests that ubiquitin mediated post translational modification is a critical regulatory process involved in diverse cellular pathways including cell death. During ubiquitination, E3 ligases recognize target proteins and...