Neta Rosenzweig
Overview
Explore the profile of Neta Rosenzweig including associated specialties, affiliations and a list of published articles.
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Articles
9
Citations
1165
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0
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Recent Articles
1.
Brandao W, Jain N, Yin Z, Kleemann K, Carpenter M, Bao X, et al.
Sci Transl Med
. 2025 Jan;
17(781):eadk3690.
PMID: 39813317
Alzheimer's disease (AD) is the most prevalent neurodegenerative disorder. Antiamyloid antibody treatments modestly slow disease progression in mild dementia due to AD. Emerging evidence shows that homeostatic dysregulation of the...
2.
Heneka M, van der Flier W, Jessen F, Hoozemanns J, Thal D, Boche D, et al.
Nat Rev Immunol
. 2024 Dec;
PMID: 39653749
Increasing evidence points to a pivotal role of immune processes in the pathogenesis of Alzheimer disease, which is the most prevalent neurodegenerative and dementia-causing disease of our time. Multiple lines...
3.
Rosenzweig N, Kleemann K, Rust T, Carpenter M, Grucci M, Aronchik M, et al.
Nat Med
. 2024 Jul;
30(10):2990-3003.
PMID: 38961225
APOE4 is the strongest genetic risk factor for Alzheimer's disease (AD), with increased odds ratios in female carriers. Targeting amyloid plaques shows modest improvement in male non-APOE4 carriers. Leveraging single-cell...
4.
Yin Z, Rosenzweig N, Kleemann K, Zhang X, Brandao W, Margeta M, et al.
Nat Immunol
. 2023 Sep;
24(11):1839-1853.
PMID: 37749326
The APOE4 allele is the strongest genetic risk factor for late-onset Alzheimer's disease (AD). The contribution of microglial APOE4 to AD pathogenesis is unknown, although APOE has the most enriched...
5.
Rosenzweig N, Dvir-Szternfeld R, Tsitsou-Kampeli A, Keren-Shaul H, Ben-Yehuda H, Weill-Raynal P, et al.
Nat Commun
. 2019 Jan;
10(1):465.
PMID: 30692527
Alzheimer's disease (AD) is a heterogeneous disorder with multiple etiologies. Harnessing the immune system by blocking the programmed cell death receptor (PD)-1 pathway in an amyloid beta mouse model was...
6.
Deczkowska A, Matcovitch-Natan O, Tsitsou-Kampeli A, Ben-Hamo S, Dvir-Szternfeld R, Spinrad A, et al.
Nat Commun
. 2017 Sep;
8(1):717.
PMID: 28959042
During ageing, microglia acquire a phenotype that may negatively affect brain function. Here we show that ageing microglial phenotype is largely imposed by interferon type I (IFN-I) chronically present in...
7.
Baruch K, Deczkowska A, Rosenzweig N, Tsitsou-Kampeli A, Sharif A, Matcovitch-Natan O, et al.
Nat Med
. 2016 Jan;
22(2):135-7.
PMID: 26779813
Systemic immune suppression may curtail the ability to mount the protective, cell-mediated immune responses that are needed for brain repair. By using mouse models of Alzheimer's disease (AD), we show...
8.
Baruch K, Rosenzweig N, Kertser A, Deczkowska A, Sharif A, Spinrad A, et al.
Nat Commun
. 2015 Aug;
6:7967.
PMID: 26284939
Alzheimer's disease (AD) is a neurodegenerative disorder in which chronic neuroinflammation contributes to disease escalation. Nevertheless, while immunosuppressive drugs have repeatedly failed in treating this disease, recruitment of myeloid cells...
9.
Kunis G, Baruch K, Rosenzweig N, Kertser A, Miller O, Berkutzki T, et al.
Brain
. 2013 Oct;
136(Pt 11):3427-40.
PMID: 24088808
Infiltrating T cells and monocyte-derived macrophages support central nervous system repair. Although infiltration of leucocytes to the injured central nervous system has recently been shown to be orchestrated by the...
10.
Shechter R, Miller O, Yovel G, Rosenzweig N, London A, Ruckh J, et al.
Immunity
. 2013 Mar;
38(3):555-69.
PMID: 23477737
Monocyte-derived macrophages are essential for recovery after spinal cord injury, but their homing mechanism is poorly understood. Here, we show that although of common origin, the homing of proinflammatory (M1)...