Neil Slaven
Overview
Explore the profile of Neil Slaven including associated specialties, affiliations and a list of published articles.
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5
Citations
96
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Recent Articles
1.
Kim Y, Kim S, Saul D, Neyazi M, Schmid M, Wakimoto H, et al.
J Clin Invest
. 2024 Dec;
135(4).
PMID: 39688912
Heterozygous truncating variants in the sarcomere protein titin (TTN) are the most common genetic cause of heart failure. To understand mechanisms that regulate abundant cardiomyocyte (CM) TTN expression, we characterized...
2.
Barozzi I, Slaven N, Canale E, Lopes R, Amorim Monteiro Barbosa I, Bleu M, et al.
Cancer Discov
. 2024 May;
14(9):1612-1630.
PMID: 38753319
Only a handful of somatic alterations have been linked to endocrine therapy resistance in hormone-dependent breast cancer, potentially explaining ∼40% of relapses. If other mechanisms underlie the evolution of hormone-dependent...
3.
Rosano D, Sofyali E, Dhiman H, Ghirardi C, Ivanoiu D, Heide T, et al.
Cancer Discov
. 2024 Mar;
14(5):866-889.
PMID: 38527495
Significance: This study advances the understanding of therapy-induced dormancy with potential clinical implications for breast cancer. Estrogen receptor-positive breast cancer cells adapt to endocrine treatment by entering a dormant state...
4.
Spurrell C, Barozzi I, Kosicki M, Mannion B, Blow M, Fukuda-Yuzawa Y, et al.
Cell Rep
. 2022 Sep;
40(12):111400.
PMID: 36130500
Heart disease is associated with re-expression of key transcription factors normally active only during prenatal development of the heart. However, the impact of this reactivation on the regulatory landscape in...
5.
Patten D, Corleone G, Gyorffy B, Perone Y, Slaven N, Barozzi I, et al.
Nat Med
. 2018 Jul;
24(9):1469-1480.
PMID: 30038216
The degree of intrinsic and interpatient phenotypic heterogeneity and its role in tumor evolution is poorly understood. Phenotypic drifts can be transmitted via inheritable transcriptional programs. Cell-type specific transcription is...