Natalia Fernandez-Borges
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Explore the profile of Natalia Fernandez-Borges including associated specialties, affiliations and a list of published articles.
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Recent Articles
11.
Fernandez-Borges N, Erana H, Castilla J
Prion
. 2018 Feb;
12(2):83-87.
PMID: 29388474
Historically, the observation of naturally occurring cases of prion disease led to the classification of different susceptibility grades and to the designation of prion resistant species. However, the development of...
12.
Sevillano A, Fernandez-Borges N, Younas N, Wang F, Elezgarai S, Bravo S, et al.
PLoS Pathog
. 2018 Feb;
14(1):e1006797.
PMID: 29385212
Very solid evidence suggests that the core of full length PrPSc is a 4-rung β-solenoid, and that individual PrPSc subunits stack to form amyloid fibers. We recently used limited proteolysis...
13.
Llorens F, Thune K, Marti E, Kanata E, Dafou D, Diaz-Lucena D, et al.
PLoS Pathog
. 2018 Jan;
14(1):e1006802.
PMID: 29357384
Increasing evidence indicates that microRNAs (miRNAs) are contributing factors to neurodegeneration. Alterations in miRNA signatures have been reported in several neurodegenerative dementias, but data in prion diseases are restricted to...
14.
Otero A, Bolea R, Hedman C, Fernandez-Borges N, Marin B, Lopez-Perez O, et al.
Mol Neurobiol
. 2017 Dec;
55(7):6182-6192.
PMID: 29264770
While prion diseases have been described in numerous species, some, including those of the Canidae family, appear to show resistance or reduced susceptibility. A better understanding of the factors underlying...
15.
Fernandez-Borges N, Parra B, Vidal E, Erana H, Sanchez-Martin M, de Castro J, et al.
PLoS Pathog
. 2017 Nov;
13(11):e1006716.
PMID: 29131852
One of the characteristics of prions is their ability to infect some species but not others and prion resistant species have been of special interest because of their potential in...
16.
Llorens F, Thune K, Tahir W, Kanata E, Diaz-Lucena D, Xanthopoulos K, et al.
Mol Neurodegener
. 2017 Nov;
12(1):83.
PMID: 29126445
Background: YKL-40 (also known as Chitinase 3-like 1) is a glycoprotein produced by inflammatory, cancer and stem cells. Its physiological role is not completely understood but YKL-40 is elevated in...
17.
Fernandez-Borges N, Di Bari M, Erana H, Sanchez-Martin M, Pirisinu L, Parra B, et al.
Acta Neuropathol
. 2017 Nov;
135(2):179-199.
PMID: 29094186
Prion diseases are caused by a misfolding of the cellular prion protein (PrP) to a pathogenic isoform named PrP. Prions exist as strains, which are characterized by specific pathological and...
18.
Erana H, Fernandez-Borges N, Elezgarai S, Harrathi C, Charco J, Chianini F, et al.
J Virol
. 2017 Oct;
91(24).
PMID: 28978705
Prion diseases, or transmissible spongiform encephalopathies (TSEs), are a group of rare progressive neurodegenerative disorders caused by an abnormally folded prion protein (PrP). This is capable of transforming the normal...
19.
Fernandez-Borges N, Erana H, Elezgarai S, Harrathi C, Venegas V, Castilla J
Methods Mol Biol
. 2017 Sep;
1658:205-216.
PMID: 28861792
Prion diseases or transmissible spongiform encephalopathies (TSEs) are a group of neurodegenerative diseases where the misfolding of the prion protein (PrP) is a crucial event. Based on studies in TSE-affected...
20.
Elezgarai S, Fernandez-Borges N, Erana H, Sevillano A, Charco J, Harrathi C, et al.
Sci Rep
. 2017 Aug;
7(1):9584.
PMID: 28851967
Human transmissible spongiform encephalopathies (TSEs) or prion diseases are a group of fatal neurodegenerative disorders that include Kuru, Creutzfeldt-Jakob disease, Gerstmann-Sträussler-Scheinker syndrome (GSS), and fatal familial insomnia. GSS is a...