Nakako Izumi Nakajima
Overview
Explore the profile of Nakako Izumi Nakajima including associated specialties, affiliations and a list of published articles.
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Articles
12
Citations
214
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Recent Articles
1.
Dose-dependent effects of histone methyltransferase NSD2 on site-specific double-strand break repair
Iwasaki K, Tojo A, Kobayashi H, Shimizu K, Kamimura Y, Horikoshi Y, et al.
Genes Cells
. 2024 Sep;
29(11):951-965.
PMID: 39245559
Histone modifications are catalyzed and recognized by specific proteins to regulate dynamic DNA metabolism processes. NSD2 is a histone H3 lysine 36 (H3K36)-specific methyltransferase that is associated with both various...
2.
Nakajima N, Yamauchi M, Kakoti S, Cuihua L, Kato R, Permata T, et al.
DNA Repair (Amst)
. 2020 Jun;
91-92:102872.
PMID: 32502756
The cell-killing effect of radiotherapy largely depends on unrepaired DNA double-stranded breaks (DSBs) or lethal chromosome aberrations induced by DSBs. Thus, the capability of DSB repair is critically important for...
3.
Iijima M, Okonogi N, Nakajima N, Morokoshi Y, Kanda H, Yamada T, et al.
J Gynecol Oncol
. 2020 Jan;
31(2):e19.
PMID: 31912675
Objective: Programmed cell death-ligand 1 (PD-L1) is expressed in tumor cells and has been shown to predict clinical outcomes of several types of malignancies. The aim of this study was...
4.
Keiko Li H, Hasegawa S, Nakajima N, Morokoshi Y, Minegishi K, Nagatsu K
Biochem Biophys Res Commun
. 2018 Nov;
506(4):1078-1084.
PMID: 30409432
Major histocompatibility complex class I chain-related protein A and B (MICA/B) are ligands of the immune receptor, natural-killer group 2 member D. MICA/B expression is often found in several types...
5.
Yasuda T, Kagawa W, Ogi T, Kato T, Suzuki T, Dohmae N, et al.
PLoS Genet
. 2018 Mar;
14(3):e1007277.
PMID: 29590107
The p300 and CBP histone acetyltransferases are recruited to DNA double-strand break (DSB) sites where they induce histone acetylation, thereby influencing the chromatin structure and DNA repair process. Whether p300/CBP...
6.
Nakajima N, Niimi A, Isono M, Oike T, Sato H, Nakano T, et al.
Oncol Rep
. 2017 Jul;
38(2):693-702.
PMID: 28677817
Immunotherapy is expected to be promising as a next generation cancer therapy. Immunoreceptors are often activated constitutively in cancer cells, however, such levels of ligand expression are not effectively recognized...
7.
Allen C, Hirakawa H, Nakajima N, Moore S, Nie J, Sharma N, et al.
Radiat Res
. 2017 May;
188(1):82-93.
PMID: 28535128
Genome instability is a hallmark of cancer cells and dysregulation or defects in DNA repair pathways cause genome instability and are linked to inherited cancer predisposition syndromes. Ionizing radiation can...
8.
Fujisawa H, Nakajima N, Sunada S, Lee Y, Hirakawa H, Yajima H, et al.
Radiat Oncol
. 2015 Aug;
10:175.
PMID: 26286029
Background: High linear energy transfer (LET) radiation such as carbon ion particles is successfully used for treatment of solid tumors. The reason why high LET radiation accomplishes greater tumor-killing than...
9.
Nakajima N, Hagiwara Y, Oike T, Okayasu R, Murakami T, Nakano T, et al.
PLoS One
. 2015 Apr;
10(3):e0122582.
PMID: 25826455
The choice of DNA double strand break (DSB) repair pathway is determined at the stage of DSB end resection. Resection was proposed to control the balance between the two major...
10.
Yajima H, Fujisawa H, Nakajima N, Hirakawa H, Jeggo P, Okayasu R, et al.
DNA Repair (Amst)
. 2013 Sep;
12(11):936-46.
PMID: 24041488
DNA double strand breaks (DSBs) induced by ionizing radiation (IR) are deleterious damages. Two major pathways repair DSBs in human cells, DNA non-homologous end-joining (NHEJ) and homologous recombination (HR). It...