Matthias Bieg
Overview
Explore the profile of Matthias Bieg including associated specialties, affiliations and a list of published articles.
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Articles
30
Citations
1493
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0
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Recent Articles
1.
Messingschlager M, Bartel-Steinbach M, Mackowiak S, Denkena J, Bieg M, Klos M, et al.
Environ Res
. 2023 Jun;
233:116413.
PMID: 37343754
While the link between exposure to high levels of ambient particulate matter (PM) and increased incidences of respiratory and cardiovascular diseases is widely recognized, recent epidemiological studies have shown that...
2.
Thurmann L, Klos M, Mackowiak S, Bieg M, Bauer T, Ishaque N, et al.
Allergy
. 2023 Jan;
78(6):1489-1506.
PMID: 36704932
Background: Childhood asthma is a result of a complex interaction of genetic and environmental components causing epigenetic and immune dysregulation, airway inflammation and impaired lung function. Although different microarray based...
3.
Hubschmann D, Kleinheinz K, Wagener R, Bernhart S, Lopez C, Toprak U, et al.
Leukemia
. 2021 May;
35(7):2002-2016.
PMID: 33953289
B cells have the unique property to somatically alter their immunoglobulin (IG) genes by V(D)J recombination, somatic hypermutation (SHM) and class-switch recombination (CSR). Aberrant targeting of these mechanisms is implicated...
4.
Haller F, Schlieben L, Ferrazzi F, Michal M, Stohr R, Moskalev E, et al.
Am J Pathol
. 2021 Apr;
191(7):1314-1324.
PMID: 33887215
Solitary fibrous tumors (SFTs) harbor activating NAB2-STAT6 gene fusions. Different variants of the NAB2-STAT6 gene fusion have been associated with distinct clinicopathologic features. Lipomatous SFTs are a morphologic variant of...
5.
Differentially methylated regions within lung cancer risk loci are enriched in deregulated enhancers
Laplana M, Bieg M, Faltus C, Melnik S, Bogatyrova O, Gu Z, et al.
Epigenetics
. 2021 Feb;
17(2):117-132.
PMID: 33595421
Genome-wide association studies (GWAS) have identified SNPs linked with lung cancer risk. Our aim was to discover the genes, non-coding RNAs, and regulatory elements within GWAS-identified risk regions that are...
6.
Bieg M, Moskalev E, Will R, Hebele S, Schwarzbach M, Schmeck S, et al.
Am J Pathol
. 2021 Jan;
191(4):602-617.
PMID: 33497701
Solitary fibrous tumors (SFTs) harbor recurrent NAB2-STAT6 gene fusions, promoting constitutional up-regulation of oncogenic early growth response 1 (EGR1)-dependent gene expression. SFTs with the most common canonical NAB2 exon 4-STAT6...
7.
Plath M, Gass J, Hlevnjak M, Li Q, Feng B, Hostench X, et al.
Int J Cancer
. 2020 Sep;
148(1):115-127.
PMID: 32930393
Genomic alterations are a driving force in the multistep process of head and neck cancer (HNC) and result from the interaction of exogenous environmental exposures and endogenous cellular processes. Each...
8.
Haller F, Moskalev E, Kuck S, Bieg M, Winkelmann C, Muller S, et al.
Am J Surg Pathol
. 2020 Apr;
44(9):1290-1292.
PMID: 32341238
No abstract available.
9.
Kim-Wanner S, Assenov Y, Nair M, Weichenhan D, Benner A, Becker N, et al.
J Thorac Oncol
. 2020 Apr;
15(8):1338-1350.
PMID: 32272161
Introduction: Surgical procedure is the treatment of choice in early stage I lung adenocarcinoma. However, a considerable number of patients experience recurrence within the first 2 years after complete resection....
10.
Feng B, Shen Y, Hostench X, Bieg M, Plath M, Ishaque N, et al.
Clin Cancer Res
. 2020 Mar;
26(14):3616-3628.
PMID: 32161122
Purpose: Malignant progression exhibits a tightly orchestrated balance between immune effector response and tolerance. However, underlying molecular principles that drive the establishment and maintenance of the tumor immune phenotype remain...