Matthew D Eldridge
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Explore the profile of Matthew D Eldridge including associated specialties, affiliations and a list of published articles.
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23
Citations
1583
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Recent Articles
11.
Bornschein J, Wernisch L, Secrier M, Miremadi A, Perner J, MacRae S, et al.
Int J Cancer
. 2019 May;
145(12):3389-3401.
PMID: 31050820
Cancers occurring at the gastroesophageal junction (GEJ) are classified as predominantly esophageal or gastric, which is often difficult to decipher. We hypothesized that the transcriptomic profile might reveal molecular subgroups...
12.
Frankell A, Jammula S, Li X, Contino G, Killcoyne S, Abbas S, et al.
Nat Genet
. 2019 Feb;
51(3):506-516.
PMID: 30718927
Esophageal adenocarcinoma (EAC) is a poor-prognosis cancer type with rapidly rising incidence. Understanding of the genetic events driving EAC development is limited, and there are few molecular biomarkers for prognostication...
13.
Mouliere F, Chandrananda D, Piskorz A, Moore E, Morris J, Ahlborn L, et al.
Sci Transl Med
. 2018 Nov;
10(466).
PMID: 30404863
Existing methods to improve detection of circulating tumor DNA (ctDNA) have focused on genomic alterations but have rarely considered the biological properties of plasma cell-free DNA (cfDNA). We hypothesized that...
14.
Harewood L, Kishore K, Eldridge M, Wingett S, Pearson D, Schoenfelder S, et al.
Genome Biol
. 2017 Jun;
18(1):125.
PMID: 28655341
Chromosomal rearrangements occur constitutionally in the general population and somatically in the majority of cancers. Detection of balanced rearrangements, such as reciprocal translocations and inversions, is troublesome, which is particularly...
15.
Staples E, Morillo-Gutierrez B, Davies J, Petersheim D, Massaad M, Slatter M, et al.
J Clin Immunol
. 2017 Apr;
37(5):415-418.
PMID: 28417298
No abstract available.
16.
Secrier M, Li X, de Silva N, Eldridge M, Contino G, Bornschein J, et al.
Nat Genet
. 2017 Feb;
49(2):317.
PMID: 28138154
No abstract available.
17.
Secrier M, Li X, de Silva N, Eldridge M, Contino G, Bornschein J, et al.
Nat Genet
. 2016 Sep;
48(10):1131-41.
PMID: 27595477
Esophageal adenocarcinoma (EAC) has a poor outcome, and targeted therapy trials have thus far been disappointing owing to a lack of robust stratification methods. Whole-genome sequencing (WGS) analysis of 129...
18.
Contino G, Eldridge M, Secrier M, Bower L, Fels Elliott R, Weaver J, et al.
F1000Res
. 2016 Sep;
5:1336.
PMID: 27594985
Esophageal adenocarcinoma (EAC) is highly mutated and molecularly heterogeneous. The number of cell lines available for study is limited and their genome has been only partially characterized. The availability of...
19.
Alioto T, Buchhalter I, Derdak S, Hutter B, Eldridge M, Hovig E, et al.
Nat Commun
. 2015 Dec;
6:10001.
PMID: 26647970
As whole-genome sequencing for cancer genome analysis becomes a clinical tool, a full understanding of the variables affecting sequencing analysis output is required. Here using tumour-normal sample pairs from two...
20.
Shaw G, Whitaker H, Corcoran M, Dunning M, Luxton H, Kay J, et al.
Eur Urol
. 2015 Nov;
70(2):214-8.
PMID: 26572708
Unlabelled: The androgen receptor (AR) is the dominant growth factor in prostate cancer (PCa). Therefore, understanding how ARs regulate the human transcriptome is of paramount importance. The early effects of...