Martina Haller
Overview
Explore the profile of Martina Haller including associated specialties, affiliations and a list of published articles.
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Articles
9
Citations
598
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Recent Articles
1.
Haller M, Khalid S, Kremser L, Fresser F, Furlan T, Hermann M, et al.
J Biol Chem
. 2016 Sep;
291(45):23557-23568.
PMID: 27624939
Dysfunctional mitochondria contribute to the development of many diseases and pathological conditions through the excessive production of reactive oxygen species (ROS), and, where studied, ablation of p66Shc (p66) was beneficial....
2.
Ichim G, Lopez J, Ahmed S, Muthalagu N, Giampazolias E, Delgado M, et al.
Mol Cell
. 2015 Feb;
57(5):860-872.
PMID: 25702873
During apoptosis, the mitochondrial outer membrane is permeabilized, leading to the release of cytochrome c that activates downstream caspases. Mitochondrial outer membrane permeabilization (MOMP) has historically been thought to occur...
3.
Haller M, Hock A, Giampazolias E, Oberst A, Green D, Debnath J, et al.
Autophagy
. 2015 Jan;
10(12):2269-78.
PMID: 25629932
During macroautophagy, conjugation of ATG12 to ATG5 is essential for LC3 lipidation and autophagosome formation. Additionally, ATG12 has ATG5-independent functions in diverse processes including mitochondrial fusion and mitochondrial-dependent apoptosis. In...
4.
Baudot A, Haller M, Mrschtik M, Tait S, Ryan K
Methods
. 2014 Dec;
75:105-11.
PMID: 25498004
Macroautophagy (hereafter termed autophagy) is a cellular membrane-trafficking process that functions to deliver cytoplasmic constituents to lysosomes for degradation. Autophagy operates at basal levels to turn over damaged and misfolded...
5.
Ashraf M, Ebner M, Wallner C, Haller M, Khalid S, Schwelberger H, et al.
Cell Commun Signal
. 2014 Jan;
12:6.
PMID: 24423080
Background: Many diseases and pathological conditions are characterized by transient or constitutive overproduction of reactive oxygen species (ROS). ROS are causal for ischemia/reperfusion (IR)-associated tissue injury (IRI), a major contributor...
6.
Tait S, Oberst A, Quarato G, Milasta S, Haller M, Wang R, et al.
Cell Rep
. 2013 Nov;
5(4):878-85.
PMID: 24268776
Programmed necrosis (or necroptosis) is a form of cell death triggered by the activation of receptor interacting protein kinase-3 (RIPK3). Several reports have implicated mitochondria and mitochondrial reactive oxygen species...
7.
Elfering A, Grebner S, Haller M
Ergonomics
. 2012 Sep;
55(12):1463-75.
PMID: 23002721
Practitioner Summary: Commuting accidents occur frequently and at high cost. This study shows that mental work demands of railway staff are related to cognitive failure and risky commuting behaviour such...
8.
Fuller T, Kusch A, Chaykovska L, Catar R, Putzer J, Haller M, et al.
Transplantation
. 2012 Aug;
94(7):679-86.
PMID: 22932117
Background: Prolonged cold preservation frequently causes delayed renal graft function resulting from tubular epithelial injury. Inhibition of signal transduction downstream from protein kinase C (PKC) may reduce renal ischemia-reperfusion injury...
9.
Kuznetsov A, Kehrer I, Kozlov A, Haller M, Redl H, Hermann M, et al.
Anal Bioanal Chem
. 2011 Feb;
400(8):2383-90.
PMID: 21336935
Reactive oxygen species (ROS) are involved in the regulation of many physiological processes. However, overproduction of ROS under various cellular stresses results in cell death and organ injury and thus...