Marie-Chloe Boulanger
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Explore the profile of Marie-Chloe Boulanger including associated specialties, affiliations and a list of published articles.
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52
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1745
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Recent Articles
11.
Rosa M, Chignon A, Li Z, Boulanger M, Arsenault B, Bosse Y, et al.
NPJ Genom Med
. 2019 Sep;
4:23.
PMID: 31552141
Growing evidence suggests that inflammation is a significant contributor to different cardiovascular diseases (CVDs). Mendelian randomization (MR) was performed to assess the causal inference between plasma soluble IL6 receptor (sIL6R),...
12.
Argaud D, Boulanger M, Chignon A, Mkannez G, Mathieu P
Nucleic Acids Res
. 2019 Jun;
47(16):8424-8438.
PMID: 31251802
ENPP2, which encodes for the enzyme autotaxin (ATX), is overexpressed during chronic inflammatory diseases and various cancers. However, the molecular mechanism involved in the ENPP2 transcription remains elusive. Here, in...
13.
Mathieu P, Boulanger M
Front Cardiovasc Med
. 2019 Mar;
6:18.
PMID: 30881959
Calcific aortic valve disease (CAVD) is a complex trait disorder characterized by calcific remodeling of leaflets. Genome-wide association (GWA) study and Mendelian randomization (MR) have highlighted that , which encodes...
14.
Bouchareb R, Boulanger M, Tastet L, Mkannez G, Nsaibia M, Hadji F, et al.
Eur Heart J
. 2018 Nov;
40(17):1362-1373.
PMID: 30395215
Aims: Calcific aortic valve stenosis (CAVS) is characterized by a fibrocalcific process. Studies have shown an association between CAVS and the activation of platelets. It is believed that shear stress...
15.
Nsaibia M, Boulanger M, Bouchareb R, Mkannez G, Dahou A, Salaun E, et al.
Clin Chim Acta
. 2018 Jul;
485:173-177.
PMID: 29966619
Introduction: Aortic valve bioprostheses, which do not mandate chronic anticoagulation, are prone to structural valve degeneration (SVD). The processes involved in SVD are likely multifactorial. We hypothesized that inflammation and...
16.
Mkannez G, Gagne-Ouellet V, Nsaibia M, Boulanger M, Rosa M, Argaud D, et al.
Cardiovasc Res
. 2018 May;
114(11):1525-1535.
PMID: 29726894
Aims: Calcific aortic valve disease (CAVD) is characterized by the osteogenic transition of valve interstitial cells (VICs). In CAVD, lysophosphatidic acid (LysoPA), a lipid mediator with potent osteogenic activity, is...
17.
Theriault S, Gaudreault N, Lamontagne M, Rosa M, Boulanger M, Messika-Zeitoun D, et al.
Nat Commun
. 2018 Mar;
9(1):988.
PMID: 29511167
Calcific aortic valve stenosis (CAVS) is a common and life-threatening heart disease and the current treatment options cannot stop or delay its progression. A GWAS on 1009 cases and 1017...
18.
Forcellini E, Boutin S, Lefebvre C, Shayhidin E, Boulanger M, Rheaume G, et al.
Eur J Med Chem
. 2018 Feb;
147:130-149.
PMID: 29427906
The ecto-nucleotide pyrophosphatase/phosphodiesterase-1 (NPP1) was recently shown to promote mineralization of the aortic valve, hence, its inhibition represents a significant target. A quinazoline-4-piperidine sulfamide compound (QPS1) has been described as...
19.
Mathieu P, Arsenault B, Boulanger M, Bosse Y, Koschinsky M
Expert Rev Cardiovasc Ther
. 2017 Aug;
15(10):797-807.
PMID: 28816078
Calcific aortic valve disease (CAVD) is the most prevalent heart valve disorder. Gene variant in the LPA gene, which encodes for apolipoprotein(a), has been associated at the genome-wide level with...
20.
Nsaibia M, Boulanger M, Bouchareb R, Mkannez G, Le Quang K, Hadji F, et al.
Cardiovasc Res
. 2017 May;
113(11):1351-1363.
PMID: 28472283
Aims: Oxidatively modified lipoproteins may promote the development/progression of calcific aortic valve stenosis (CAVS). Oxidative transformation of low-density lipoprotein (OxLDL) generates lysophosphatidic acid (LPA), a lipid mediator that accumulates in...