Lior Zilberberg
Overview
Explore the profile of Lior Zilberberg including associated specialties, affiliations and a list of published articles.
Author names and details appear as published. Due to indexing inconsistencies, multiple individuals may share a name, and a single author may have variations. MedLuna displays this data as publicly available, without modification or verification
Snapshot
Snapshot
Articles
21
Citations
1017
Followers
0
Related Specialties
Related Specialties
Top 10 Co-Authors
Top 10 Co-Authors
Published In
Published In
Affiliations
Affiliations
Soon will be listed here.
Recent Articles
1.
Saddic L, Kaneda G, Momenzadeh A, Zilberberg L, Song Y, Mastali M, et al.
bioRxiv
. 2025 Mar;
PMID: 40027651
Background: Single-cell omics technology is a powerful tool in biomedical research. However, single cell proteomics has lagged due to an inability to amplify peptides in a similar fashion to nucleotide...
2.
Sachan N, Phoon C, Bu L, Zilberberg L, Ahamed J, Rifkin D
Matrix Biol Plus
. 2024 Jun;
22:100149.
PMID: 38831847
Although the mechanism for activation of latent TGFβ1 and TGFβ3 is understood to involve the binding of the TGFβ propeptide (LAP) to both an integrin and an insoluble substrate, the...
3.
Saddic L, Escopete S, Zilberberg L, Kalsow S, Gupta D, Eghbali M, et al.
Int J Mol Sci
. 2023 Sep;
24(17).
PMID: 37686377
Marfan syndrome causes a hereditary form of thoracic aortic aneurysms with worse outcomes in male compared to female patients. In this study, we examine the effects of 17 β-estradiol on...
4.
Sachan N, Phoon C, Zilberberg L, Kugler M, Ene T, Mintz S, et al.
Matrix Biol
. 2023 May;
121:41-55.
PMID: 37217119
To assess the contribution of individual TGF-β isoforms to aortopathy in Marfan syndrome (MFS), we quantified the survival and phenotypes of mice with a combined fibrillin1 (the gene defective in...
5.
Saddic L, Escopete S, Zilberberg L, Kalsow S, Gupta D, Egbhali M, et al.
bioRxiv
. 2023 May;
PMID: 37215011
Marfan syndrome causes a hereditary form of thoracic aortic aneurysms with dilation of the aortic root. Human and animal models suggest a worse phenotype for males compared to females with...
6.
Guo D, Regalado E, Pinard A, Chen J, Lee K, Rigelsky C, et al.
Am J Hum Genet
. 2018 Apr;
102(4):706-712.
PMID: 29625025
The major diseases affecting the thoracic aorta are aneurysms and acute dissections, and pathogenic variants in 11 genes are confirmed to lead to heritable thoracic aortic disease. However, many families...
7.
Deryugina E, Zajac E, Zilberberg L, Muramatsu T, Joshi G, Dabovic B, et al.
Oncogene
. 2018 Jan;
37(14):1815-1829.
PMID: 29348457
Latent transforming growth factor β (TGFβ)-binding proteins (LTBPs) are important for the secretion, activation, and function of mature TGFβ, especially so in cancer cell physiology. However, specific roles of the...
8.
Rifkin D, Rifkin W, Zilberberg L
Matrix Biol
. 2017 Dec;
71-72:90-99.
PMID: 29217273
The latent transforming growth factor (TGF) β binding proteins (LTBP) are crucial mediators of TGFβ function, as they control growth factor secretion, matrix deposition, presentation and activation. Deficiencies in specific...
9.
Zilberberg L, Phoon C, Robertson I, Dabovic B, Ramirez F, Rifkin D
Proc Natl Acad Sci U S A
. 2015 Oct;
112(45):14012-7.
PMID: 26494287
Marfan syndrome (MFS) is an autosomal dominant disorder of connective tissue, caused by mutations of the microfibrillar protein fibrillin-1, that predisposes affected individuals to aortic aneurysm and rupture and is...
10.
Robertson I, Horiguchi M, Zilberberg L, Dabovic B, Hadjiolova K, Rifkin D
Matrix Biol
. 2015 May;
47:44-53.
PMID: 25960419
The LTBPs (or latent transforming growth factor β binding proteins) are important components of the extracellular matrix (ECM) that interact with fibrillin microfibrils and have a number of different roles...