K L Bennett
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Explore the profile of K L Bennett including associated specialties, affiliations and a list of published articles.
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49
Citations
1373
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Recent Articles
1.
Caputo B, De Marco C, Pichler V, Botta G, Bennett K, Clarkson C, et al.
Res Sq
. 2024 Apr;
PMID: 38562903
The two main Afrotropical malaria vectors - and - are genetically distinct and reproductively isolated across West Africa. However, populations at the western extreme of their range are assigned as...
2.
Perego M, Maurer M, Wang J, Shaffer S, Muller A, Parapatics K, et al.
Oncogene
. 2017 Sep;
37(3):302-312.
PMID: 28925403
Melanoma is a heterogeneous tumor with different subpopulations showing different proliferation rates. Slow-cycling cells were previously identified in melanoma, but not fully biologically characterized. Using the label-retention method, we identified...
3.
Peter B, Winter G, Blatt K, Bennett K, Stefanzl G, Rix U, et al.
Leukemia
. 2015 Sep;
30(2):464-72.
PMID: 26349526
Proteomic-based drug testing is an emerging approach to establish the clinical value and anti-neoplastic potential of multikinase inhibitors. The multikinase inhibitor midostaurin (PKC412) is a promising new agent used to...
4.
Fauster A, Rebsamen M, Huber K, Bigenzahn J, Stukalov A, Lardeau C, et al.
Cell Death Dis
. 2015 May;
6:e1767.
PMID: 25996294
Necroptosis is a form of regulated necrotic cell death mediated by receptor-interacting serine/threonine-protein kinase 1 (RIPK1) and RIPK3. Necroptotic cell death contributes to the pathophysiology of several disorders involving tissue...
5.
Borgdorff V, Rix U, Winter G, Gridling M, Muller A, Breitwieser F, et al.
Oncogene
. 2013 Jun;
33(19):2531-9.
PMID: 23728343
The microphthalmia-associated transcription factor (MITF) is indispensable for the viability of melanocytic cells, is an oncogene in melanoma and has a cell type-specific expression pattern. As the modulation of MITF...
6.
Rix U, Remsing Rix L, Terker A, Fernbach N, Hantschel O, Planyavsky M, et al.
Leukemia
. 2009 Nov;
24(1):44-50.
PMID: 19890374
Resistance to the BCR-ABL tyrosine kinase inhibitor imatinib poses a pressing challenge in treating chronic myeloid leukemia (CML). This resistance is often caused by point mutations in the ABL kinase...
7.
Remsing Rix L, Rix U, Colinge J, Hantschel O, Bennett K, Stranzl T, et al.
Leukemia
. 2008 Nov;
23(3):477-85.
PMID: 19039322
The detailed molecular mechanism of action of second-generation BCR-ABL tyrosine kinase inhibitors, including perturbed targets and pathways, should contribute to rationalized therapy in chronic myeloid leukemia (CML) or in other...
8.
Gangestad S, Bennett K, Thornhill R
Proc Biol Sci
. 2001 Aug;
268(1477):1677-84.
PMID: 11506680
A single trait's fluctuating asymmetry (FA) is expected to be a poor measure of developmental instability. Hence, studies that examine associations between FA and outcomes expected to covary with developmental...
9.
Bennett K, Kussmann M, Bjork P, Godzwon M, Mikkelsen M, Sorensen P, et al.
Protein Sci
. 2000 Sep;
9(8):1503-18.
PMID: 10975572
The intermolecular contact regions between monomers of the homodimeric DNA binding protein ParR and the interaction between the glycoproteins CD28 and CD80 were investigated using a strategy that combined chemical...
10.
Bennett K, Matthiesen T, Roepstorff P
Methods Mol Biol
. 2000 Aug;
146:113-31.
PMID: 10948499
No abstract available.