Jerid W Robinson
Overview
Explore the profile of Jerid W Robinson including associated specialties, affiliations and a list of published articles.
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Articles
19
Citations
345
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0
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Recent Articles
1.
Wagner B, Robinson J, Prickett T, Espiner E, Khosla S, Gaddy D, et al.
Calcif Tissue Int
. 2022 Aug;
111(5):506-518.
PMID: 35947145
C-type natriuretic peptide (CNP) activation of guanylyl cyclase-B (GC-B) catalyzes the synthesis of cGMP in chondrocytes and osteoblasts. Elevated cGMP stimulates long bone growth, and inactivating mutations in CNP or...
2.
Wagner B, Robinson J, Healy C, Gauthier M, Dickey D, Yee S, et al.
FASEB J
. 2021 Dec;
36(1):e22069.
PMID: 34859913
Atrial natriuretic peptide (NP) and BNP increase cGMP, which reduces blood pressure and cardiac hypertrophy by activating guanylyl cyclase (GC)-A, also known as NPR-A or Npr1. Although GC-A is highly...
3.
Wagner B, Robinson J, Lin Y, Lee Y, Kaci N, Legeai-Mallet L, et al.
JCI Insight
. 2021 Mar;
6(9).
PMID: 33784257
Activating mutations in the fibroblast growth factor receptor 3 (FGFR3) or inactivating mutations in guanylyl cyclase-B (GC-B), also known as NPR-B or Npr2, cause short-limbed dwarfism. FGFR3 activation causes dephosphorylation...
4.
Egbert J, Robinson J, Uliasz T, Potter L, Jaffe L
Biol Reprod
. 2021 Feb;
104(5):939-941.
PMID: 33624759
No abstract available.
5.
Robinson J, Blixt N, Norton A, Mansky K, Ye Z, Aparicio C, et al.
Bone
. 2020 Mar;
135:115320.
PMID: 32179168
C-type natriuretic peptide (CNP) activation of guanylyl cyclase (GC)-B, also known as NPR2, stimulates cGMP synthesis and bone elongation. CNP activation requires the phosphorylation of multiple GC-B residues and dephosphorylation...
6.
Schmidt H, Dickey D, Dumoulin A, Octave M, Robinson J, Kuhn R, et al.
J Neurosci
. 2018 Sep;
38(45):9768-9780.
PMID: 30249793
cGMP signaling elicited by activation of the transmembrane receptor guanylyl cyclase Npr2 (also known as guanylyl cyclase B) by the ligand CNP controls sensory axon bifurcation of DRG and cranial...
7.
Shuhaibar L, Robinson J, Vigone G, Shuhaibar N, Egbert J, Baena V, et al.
Elife
. 2017 Dec;
6.
PMID: 29199951
Activating mutations in fibroblast growth factor (FGF) receptor 3 and inactivating mutations in the NPR2 guanylyl cyclase both cause severe short stature, but how these two signaling systems interact to...
8.
Robinson J, Egbert J, Davydova J, Schmidt H, Jaffe L, Potter L
Cell Signal
. 2017 Oct;
40:222-229.
PMID: 28964968
Activating mutations in fibroblast growth factor receptor 3 (FGFR3) and inactivating mutations of guanylyl cyclase-B (GC-B, also called NPRB or NPR2) cause dwarfism. FGF exposure inhibits GC-B activity in a...
9.
Dickey D, Edmund A, Otto N, Chaffee T, Robinson J, Potter L
J Biol Chem
. 2016 Mar;
291(21):11385-93.
PMID: 26980729
C-type natriuretic peptide activation of guanylyl cyclase B (GC-B), also known as natriuretic peptide receptor B or NPR2, stimulates long bone growth, and missense mutations in GC-B cause dwarfism. Four...
10.
Wang S, Jacobsen C, Carmichael H, Edmund A, Robinson J, Olney R, et al.
Hum Mutat
. 2015 Feb;
36(4):474-81.
PMID: 25703509
Based on the observation of reduced stature in relatives of patients with acromesomelic dysplasia, Maroteaux type (AMDM), caused by homozygous or compound heterozygous mutations in natriuretic peptide receptor-B gene (NPR2),...