Jeffrey J Olearczyk
Overview
Explore the profile of Jeffrey J Olearczyk including associated specialties, affiliations and a list of published articles.
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Articles
9
Citations
439
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Recent Articles
1.
Olearczyk J, Quigley J, Mitchell B, Yamamoto T, Kim I, Newman J, et al.
Clin Sci (Lond)
. 2008 May;
116(1):61-70.
PMID: 18459944
Hypertension and Type 2 diabetes are co-morbid diseases that lead to the development of nephropathy. sEH (soluble epoxide hydrolase) inhibitors are reported to provide protection from renal injury. We hypothesized...
2.
Said N, Socha M, Olearczyk J, Elmarakby A, Imig J, Motamed K
Mol Cancer Res
. 2007 Oct;
5(10):1015-30.
PMID: 17951402
Malignant ascites is a major source of morbidity and mortality in ovarian cancer patients. It functions as a permissive reactive tumor-host microenvironment and provides sustenance for the floating tumor cells...
3.
Elmarakby A, Quigley J, Olearczyk J, Sridhar A, Cook A, Inscho E, et al.
Hypertension
. 2007 Oct;
50(6):1069-76.
PMID: 17938380
The present study was designed to determine whether chemokine receptor 2b (CCR2b) contributes to the development of renal injury in salt-sensitive angiotensin II (ANG) hypertension. Rats were infused with ANG...
4.
Olearczyk J, Field M, Kim I, Morisseau C, Hammock B, Imig J
J Pharmacol Exp Ther
. 2006 Jun;
318(3):1307-14.
PMID: 16772540
The epoxyeicosatrienoic acids (EETs) have been identified as endothelium-derived hyperpolarizing factors. Metabolism of the EETs to the dihydroxyeicosatrienoic acids is catalyzed by soluble epoxide hydrolase (sEH). Administration of urea-based sEH...
5.
Imig J, Zhao X, Zaharis C, Olearczyk J, Pollock D, Newman J, et al.
Hypertension
. 2005 Sep;
46(4):975-81.
PMID: 16157792
The present study tested the hypothesis that increasing epoxyeicosatrienoic acids by inhibition of soluble epoxide hydrolase (sEH) would lower blood pressure and ameliorate renal damage in salt-sensitive hypertension. Rats were...
6.
Olearczyk J, Stephenson A, Lonigro A, Sprague R
Am J Physiol Heart Circ Physiol
. 2004 Apr;
287(2):H748-54.
PMID: 15072952
The release of ATP from erythrocytes involves a signal transduction pathway of which cystic fibrosis transmembrane conductance regulator, PKA, adenylyl cyclase, and the heterotrimeric G proteins G(s) and G(i) are...
7.
Olearczyk J, Ellsworth M, Stephenson A, Lonigro A, Sprague R
J Pharmacol Exp Ther
. 2004 Feb;
309(3):1079-84.
PMID: 14766946
Erythrocytes have been reported to release ATP from intracellular stores into the surrounding environment in response to decreased oxygen tension and mechanical deformation. This erythrocyte-derived ATP can then act on...
8.
Olearczyk J, Stephenson A, Lonigro A, Sprague R
Am J Physiol Heart Circ Physiol
. 2003 Nov;
286(3):H940-5.
PMID: 14615280
Erythrocytes are reported to release ATP in response to mechanical deformation and decreased oxygen tension. Previously we proposed that receptor-mediated activation of the heterotrimeric G protein G(s) resulted in ATP...
9.
Sprague R, Olearczyk J, Spence D, Stephenson A, Sprung R, Lonigro A
Am J Physiol Heart Circ Physiol
. 2003 Apr;
285(2):H693-700.
PMID: 12689860
Previously, it was reported that red blood cells (RBCs) are required to demonstrate participation of nitric oxide (NO) in the regulation of rabbit pulmonary vascular resistance (PVR). RBCs do not...