Ioannis Dragatsis
Overview
Explore the profile of Ioannis Dragatsis including associated specialties, affiliations and a list of published articles.
Author names and details appear as published. Due to indexing inconsistencies, multiple individuals may share a name, and a single author may have variations. MedLuna displays this data as publicly available, without modification or verification
Snapshot
Snapshot
Articles
46
Citations
2665
Followers
0
Related Specialties
Related Specialties
Top 10 Co-Authors
Top 10 Co-Authors
Published In
Published In
Affiliations
Affiliations
Soon will be listed here.
Recent Articles
1.
Saito-Diaz K, Dietrich P, Saini T, Rashid M, Wu H, Ishan M, et al.
Sci Transl Med
. 2024 Nov;
16(774):eadq2418.
PMID: 39565876
The peripheral nervous system (PNS) is essential for proper body function. A high percentage of the world's population suffers from nerve degeneration or peripheral nerve damage. Despite this, there are...
2.
Saito-Diaz K, Dietrich P, Wu H, Sun X, Patel A, Wzientek C, et al.
bioRxiv
. 2023 Mar;
PMID: 36993570
The peripheral nervous system (PNS) is essential for proper body function. A high percentage of the population suffer nerve degeneration or peripheral damage. For example, over 40% of patients with...
3.
Dietrich P, Dragatsis I
Neural Regen Res
. 2022 Aug;
18(3):523-524.
PMID: 36018159
No abstract available.
4.
Chekuri A, Logan E, Krauson A, Salani M, Ackerman S, Kirchner E, et al.
Hum Mol Genet
. 2021 Dec;
31(11):1776-1787.
PMID: 34908112
Familial dysautonomia (FD) is an autosomal recessive neurodegenerative disease caused by a splicing mutation in the gene encoding Elongator complex protein 1 (ELP1, also known as IKBKAP). This mutation results...
5.
Dietrich P, Alli S, Mulligan M, Cox R, Ashbrook D, Williams R, et al.
Neurobiol Dis
. 2021 Dec;
162:105581.
PMID: 34871739
Mitochondria dysfunction occurs in the aging brain as well as in several neurodegenerative disorders and predisposes neuronal cells to enhanced sensitivity to neurotoxins. 3-nitropropionic acid (3-NP) is a naturally occurring...
6.
Even A, Morelli G, Turchetto S, Shilian M, Le Bail R, Laguesse S, et al.
Nat Commun
. 2021 Nov;
12(1):6678.
PMID: 34772940
No abstract available.
7.
Even A, Morelli G, Turchetto S, Shilian M, Le Bail R, Laguesse S, et al.
Nat Commun
. 2021 Oct;
12(1):5878.
PMID: 34620845
Microtubule (MT)-based transport is an evolutionary conserved process finely tuned by posttranslational modifications. Among them, α-tubulin acetylation, primarily catalyzed by a vesicular pool of α-tubulin N-acetyltransferase 1 (Atat1), promotes the...
8.
Poplawski G, Kawaguchi R, Van Niekerk E, Lu P, Mehta N, Canete P, et al.
Nature
. 2020 May;
581(7806):77-82.
PMID: 32376949
Grafts of spinal-cord-derived neural progenitor cells (NPCs) enable the robust regeneration of corticospinal axons and restore forelimb function after spinal cord injury; however, the molecular mechanisms that underlie this regeneration...
9.
Burrus C, McKinstry S, Kim N, Ozlu M, Santoki A, Fang F, et al.
Cell Rep
. 2020 Jan;
30(3):642-657.e6.
PMID: 31968243
Huntington's disease (HD) is caused by an autosomal dominant polyglutamine expansion mutation of Huntingtin (HTT). HD patients suffer from progressive motor, cognitive, and psychiatric impairments, along with significant degeneration of...
10.
Fox L, Kim K, Johnson C, Chen S, Croce K, Victor M, et al.
Neuron
. 2020 Jan;
105(5):813-821.e6.
PMID: 31899071
Despite being an autosomal dominant disorder caused by a known coding mutation in the gene HTT, Huntington's disease (HD) patients with similar trinucleotide repeat mutations can have an age of...