Felicidad Almazan
Overview
Explore the profile of Felicidad Almazan including associated specialties, affiliations and a list of published articles.
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Articles
18
Citations
1104
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0
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Recent Articles
1.
Mao R, Meng S, Gu Q, Araujo-Gutierrez R, Kumar S, Yan Q, et al.
Circ Res
. 2017 Mar;
120(11):1727-1739.
PMID: 28325782
Rationale: Angiogenesis improves perfusion to the ischemic tissue after acute vascular obstruction. Angiogenesis in pathophysiological settings reactivates signaling pathways involved in developmental angiogenesis. We showed previously that AIBP (apolipoprotein A-I...
2.
Choi S, Gonen A, Diehl C, Kim J, Almazan F, Witztum J, et al.
Autophagy
. 2015 May;
11(5):785-95.
PMID: 25946330
Adaptive immunity, which plays an important role in the development of atherosclerosis, is mediated by major histocompatibility complex (MHC)-dependent antigen presentation. In atherosclerotic lesions, macrophages constitute an important class of...
3.
Ai D, Jiang H, Westerterp M, Murphy A, Wang M, Ganda A, et al.
Circ Res
. 2014 Apr;
114(10):1576-84.
PMID: 24687132
Rationale: The mammalian target of rapamycin complex 1 inhibitor, rapamycin, has been shown to decrease atherosclerosis, even while increasing plasma low-density lipoprotein levels. This suggests an antiatherogenic effect possibly mediated...
4.
Choi S, Yin H, Ravandi A, Armando A, Dumlao D, Kim J, et al.
PLoS One
. 2013 Dec;
8(12):e83145.
PMID: 24376657
Oxidation of low-density lipoprotein (LDL) is one of the major causative mechanisms in the development of atherosclerosis. In previous studies, we showed that minimally oxidized LDL (mmLDL) induced inflammatory responses...
5.
Montano E, Boullier A, Almazan F, Binder C, Witztum J, Hartvigsen K
J Lipid Res
. 2013 Sep;
54(11):3206-14.
PMID: 23997238
Macrophages play a key role in atherogenesis in part through excessive uptake of oxidized LDL (OxLDL) via scavenger receptors. Binding of OxLDL to macrophages has traditionally been assessed using radiolabeled...
6.
Fang L, Choi S, Baek J, Liu C, Almazan F, Ulrich F, et al.
Nature
. 2013 May;
498(7452):118-22.
PMID: 23719382
Cholesterol is a structural component of the cell and is indispensable for normal cellular function, although its excess often leads to abnormal proliferation, migration, inflammatory responses and/or cell death. To...
7.
Wiesner P, Tafelmeier M, Chittka D, Choi S, Zhang L, Byun Y, et al.
J Lipid Res
. 2013 May;
54(7):1877-83.
PMID: 23667177
Lipoprotein oxidation plays an important role in pathogenesis of atherosclerosis. Oxidized low density lipoprotein (OxLDL) induces profound inflammatory responses in vascular cells, such as production of monocyte chemoattractant protein-1 (MCP-1)...
8.
Choi S, Wiesner P, Almazan F, Kim J, Miller Y
PLoS One
. 2012 Mar;
7(2):e32378.
PMID: 22384232
Oxidative modification of low-density lipoprotein (LDL) turns it into an endogenous ligand recognized by pattern-recognition receptors. We have demonstrated that minimally oxidized LDL (mmLDL) binds to CD14 and mediates TLR4/MD-2-dependent...
9.
Fang L, Harkewicz R, Hartvigsen K, Wiesner P, Choi S, Almazan F, et al.
J Biol Chem
. 2010 Aug;
285(42):32343-51.
PMID: 20710028
A novel hypercholesterolemic zebrafish model has been developed to study early events of atherogenesis. This model utilizes optically transparent zebrafish larvae, fed a high cholesterol diet (HCD), to monitor processes...
10.
Wiesner P, Choi S, Almazan F, Benner C, Huang W, Diehl C, et al.
Circ Res
. 2010 May;
107(1):56-65.
PMID: 20489162
Rationale: Oxidized low-density lipoprotein (LDL) is an important determinant of inflammation in atherosclerotic lesions. It has also been documented that certain chronic infectious diseases, such as periodontitis and chlamydial infection,...