Eva B Brittebo
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Explore the profile of Eva B Brittebo including associated specialties, affiliations and a list of published articles.
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19
Citations
356
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Recent Articles
1.
Karlsson O, Jiang L, Ersson L, Malmstrom T, Ilag L, Brittebo E
Sci Rep
. 2015 Oct;
5:15570.
PMID: 26498001
β-Methylamino-L-alanine (BMAA) is implicated in the aetiology of neurodegenerative disorders. Neonatal exposure to BMAA induces cognitive impairments and progressive neurodegenerative changes including intracellular fibril formation in the hippocampus of adult...
2.
Andersson M, Karlsson O, Bergstrom U, Brittebo E, Brandt I
PLoS One
. 2015 Jul;
10(7):e0133110.
PMID: 26172384
No abstract available.
3.
Karlsson O, Berg A, Hanrieder J, Arnerup G, Lindstrom A, Brittebo E
Arch Toxicol
. 2014 May;
89(3):423-36.
PMID: 24798087
The environmental neurotoxin β-N-methylamino-L-alanine (BMAA) has been implicated in the etiology of neurodegenerative disease, and recent studies indicate that BMAA can be misincorporated into proteins. BMAA is a developmental neurotoxicant...
4.
Hanrieder J, Gerber L, Persson Sandelius A, Brittebo E, Ewing A, Karlsson O
ACS Chem Neurosci
. 2014 May;
5(7):568-75.
PMID: 24779349
The environmental neurotoxin β-N-methylamino-L-alanine (BMAA) is suggested to be linked with neurodegenerative disease. In a rat model, neonatal exposure to BMAA induced selective uptake in the hippocampus and caused cell...
5.
Karlsson O, Jiang L, Andersson M, Ilag L, Brittebo E
Toxicol Lett
. 2014 Jan;
226(1):1-5.
PMID: 24472610
The environmental neurotoxin β-N-methylamino-L-alanine (BMAA) is not an amino acid that is normally found in proteins. Our previous autoradiographic study of (3)H-labeled BMAA in adult mice unexpectedly revealed a tissue...
6.
Andersson M, Karlsson O, Bergstrom U, Brittebo E, Brandt I
PLoS One
. 2013 Nov;
8(10):e78133.
PMID: 24194910
The cyanobacterial neurotoxin β-N-methylamino-L-alanine (BMAA) has been implicated in the etiology of neurodegenerative disease and proposed to be biomagnified in terrestrial and aquatic food chains. We have previously shown that...
7.
Karlsson O, Kultima K, Wadensten H, Nilsson A, Roman E, Andren P, et al.
J Proteome Res
. 2013 Feb;
12(4):1678-90.
PMID: 23410195
The cyanobacterial toxin β-N-methylamino-l-alanine (BMAA) is suggested to play a role in neurodegenerative disease. We have previously shown that although the selective uptake of BMAA in the rodent neonatal striatum...
8.
Karlsson O, Berg A, Lindstrom A, Hanrieder J, Arnerup G, Roman E, et al.
Toxicol Sci
. 2012 Aug;
130(2):391-404.
PMID: 22872059
The cyanobacterial toxin β-N-methylamino-L-alanine (BMAA) has been proposed to contribute to neurodegenerative disease. We have previously reported a selective uptake of BMAA in the mouse neonatal hippocampus and that exposure...
9.
Karlsson O, Roman E, Berg A, Brittebo E
Behav Brain Res
. 2011 Feb;
219(2):310-20.
PMID: 21315110
We have reported previously that exposure to the cyanobacterial neurotoxin β-N-methylamino-L-alanine (BMAA) during the neonatal period causes cognitive impairments in adult rats. The aim of this study was to investigate...
10.
Karlsson O, Roman E, Brittebo E
Toxicol Sci
. 2009 Aug;
112(1):185-95.
PMID: 19692667
Most cyanobacteria (blue-green algae) can produce the neurotoxin beta-N-methylamino-L-alanine (BMAA). Dietary exposure to BMAA has been suggested to be involved in the etiology of the neurodegenerative disease amyotrophic lateral sclerosis/Parkinsonism-dementia...